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Cytokeratin 19 and cytokeratin 15 are key markers for monitoring the quality and self-renewing potential of engineered skin.

Skin stem cells maintain and repair the outer layer of skin, with some types being essential for healing wounds.

Different markers are found in stem cells of the scalp's hair follicle bulge and the surrounding skin.

The method allows for 3D tracking of hair follicle stem cells and shows they can regenerate hair for up to 180 days.

Environmental cues can change the fate and function of epithelial cells, with potential for cell therapy.

Skin cell types develop when specific genes are turned on by removing certain chemical tags from DNA.

Basal cell carcinoma mostly starts from cells in the upper skin layers, not hair follicle stem cells.

Scientists turned mouse stem cells into skin cells that can grow into skin layers and structures.

Cell-based therapies using dermal papilla cells and adipocyte lineage cells show potential for hair regeneration.

Certain genetic markers linked to reproductive potential were identified by their impact on a protein's ability to bind to genes.

Different body parts have varying levels of certain hair follicle markers.

Removing a specific gene in certain skin cells causes hair loss on the body by disrupting normal hair development.

Telogen is an active phase with important biological processes, not a resting phase.

Certain skin cells near the base of hair muscles may help renew and stabilize skin, possibly affecting skin disorder understanding.

Researchers identified potential markers for human hair color stem cells.

Skin lesions in Carney complex are likely caused by a specific group of skin cells that promote pigment production due to a genetic mutation.

Super-enhancers controlled by pioneer factors like SOX9 are crucial for stem cell adaptability and identity.

Certain proteins, Lgr5 and Lgr6, are important markers of adult stem cells and are involved in tissue repair and cancer development.

Hair follicle stem cells use specific chromatin changes to control their growth and differentiation.

Activating β-catenin in different skin stem cells causes various types of hair growth and skin tumors.

Adult esophageal cells can start to become like skin cells, with a key pathway influencing this change.

Activating β-catenin increases melanocytes and decreases Schwann cells.

Androgen is important in controlling stem cell differentiation, reducing fat development, and increasing lean mass.

Markers CRABP1, Nestin, and Ephrin B2 are present in skin cancer environments and may influence their development.

Lack of Ctip2 in skin cells delays wound healing and disrupts hair follicle stem cell markers in mice.

Laser treatment and synovial fluid can change hair follicle cells to resemble joint cells, with the changes being more significant when both treatments are used together.

Removing Mediator 1 from certain mouse cells causes teeth to grow hair instead of enamel.

Forensic DNA Phenotyping can help predict physical traits from crime scene DNA to identify suspects.

Certain signals are important for reducing specific chemical markers on hair follicle stem cells during rest periods, which is necessary for healthy hair growth.

The enzymes Tet2 and Tet3 are important for skin cell development and hair growth.