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Overactivating Hedgehog signaling makes hair follicle cells in mice grow hair faster and create more follicles.

Healing of heart and skin wounds in animals are similar.

Sox21 is crucial for tooth development and enamel formation by preventing cells from changing into a different type.

Genetic changes in mice help understand skin and hair disorders, aiding treatment development for acne and hair loss.

Follistatin and LIN28B together improve the ability of inner ear cells in mice to regenerate into hearing cells.

Different species use the same genes for tooth regeneration.

Mice without certain skin enzymes have faster hair growth and bigger eye glands.

Some skin tumors may start from hair follicle stem cells.

The study found key factors in the cause of hidradenitis suppurativa, its link to other diseases, and identified existing drugs that could potentially treat it.

Epidermal stem cells create and maintain skin structures like hair and nails through specific signaling pathways and vary by location and function.

Activin A and follistatin control when hair cells develop in mouse ears.

Key genes crucial for sheep hair follicle development were identified, aiding fine wool breeding and human hair loss research.

The genes KRT25 and SP6 affect curly hair in horses, with KRT25 also causing hair loss. If both genes are mutated, the horse gets curly hair and hair loss. KRT25 can hide the effect of SP6.

Scientists created a functional 3D skin system from stem cells that can be transplanted into wounds.

Cyclosporin extends hair growth in mice, but high-dose corticosteroids block this effect.

Blocking certain immune signals can reduce skin damage from radiation therapy.

Intermediate filaments are crucial for cell differentiation and stem cell function.

Certain genes and pathways are linked to the production of finer and denser wool in Hetian sheep.

GDNF helps grow hair and heal skin wounds by acting on hair stem cells.

UV exposure reduces Lgr6+ stem cells in mouse skin and they don't significantly contribute to skin cancer development.

RSPO1 mutations in certain patients lead to skin cells that don't develop properly and are more likely to become invasive, increasing the risk of skin cancer.

Eyelid cells share signaling components but differ in pathway activity.

Overactive Wnt signaling in mouse skin stem cells causes acne-like cysts and shrinking oil glands, which some treatments can partially fix.

White blood cells and their traps can slow down the process of new hair growth after a wound.

The research showed how melanocytes develop, move, and respond to UV light, and their stem cells' role in hair color and skin cancer risk.

Your genes, especially IL-1a, can predict how well a hair growth treatment with platelet rich plasma will work for you.

The enzymes Tet2 and Tet3 are important for skin cell development and hair growth.

HSPGs help control stem cell behavior, affecting hair growth and offering a target for hair loss treatments.

Ptch2 plays a key role in controlling stem cell function and the ability to regenerate after birth.

A topical BRAF inhibitor, vemurafenib, can speed up wound healing and promote hair growth, especially in diabetic patients.