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New insights into cell communication in psoriasis suggest innovative drug treatments.

The gene Ascl4 is not necessary for the development of hair, teeth, or mammary glands.

A specific enzyme is essential for proper hair follicle stem cell development and healthy skin.

Aged individuals heal wounds less effectively due to specific immune cell issues.

Skin spheroids with both outer and inner layers are key for regrowing skin patterns and hair.

Keratinocytes and adipose-derived stem cells can effectively heal difficult skin wounds.

Too much β-catenin activity can mess up the development of mammary glands and make them more like hair follicles.

Certain long non-coding RNAs are important for controlling hair growth cycles in sheep.

The article concludes that understanding the molecular processes in hair follicle development can improve the quality of fibers like Angora and cashmere.

Shi-Bi-Man activates hair follicle stem cells and promotes hair growth by changing lactic acid metabolism and other cellular processes.

Sweat gland development involves two unique skin cell programs and a temporary skin environment.

IL-33 is linked to hair follicle damage in psoriasis and could be a treatment target for hair loss in this condition.

Blocking IL-17 can reduce skin inflammation in a mouse model of pityriasis rubra pilaris.

MicroRNA-205 helps hair grow by changing the stiffness and contraction of hair follicle cells.

Understanding genetics is crucial for treating heart and skin diseases.

Primary cilia affect the size and oil production of eye glands but not the oil's makeup.

Skin lesions in Carney complex are likely caused by a specific group of skin cells that promote pigment production due to a genetic mutation.

Melatonin affects certain genes and pathways involved in cashmere goat hair growth.

Researchers found a genetic link for hereditary hair loss but need more analysis to identify the exact gene.

Melatonin can increase cashmere yield by altering gene expression and restarting the growth cycle early.

A specific immune response helps control mite populations on the skin, maintaining healthy hair follicles.

Stress can cause a type of hair loss in mice lacking the CCHCR1 gene.

The enzymes Tet1, Tet2, and Tet3 are important for the development of hair follicles and determining hair shape by controlling hair keratin genes.

The HoxC gene cluster and its enhancers are essential for developing hair and nails in mammals.

Researchers found four key stages of cell development that are important for hair growth and shedding in cashmere goats.

A 37-year-old male with severe skin and internal issues has a rare inherited skin condition called dystrophic epidermolysis bullosa.

The 3D-SeboSkin model effectively simulates Hidradenitis suppurativa and is useful for future research.

Dermal Papilla Cells grown in 3D and with stem cells better mimic natural hair growth conditions than cells grown in 2D.

Alopecia in patients with epidermolysis bullosa varies in severity and is often caused by skin blistering or trauma.

The gene Prss53 affects hair shape and bone development in rabbits.