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Overexpression of sPLA2-IIA in mouse skin reduces hair stem cells and increases cell differentiation through JNK/c-Jun pathway activation.

A specific chemical change in the S100A3 protein leads to the formation of a four-part structure important for hair formation.

Prolactin may play a significant role in skin and hair health and could be a target for treating skin and hair disorders.

Epithelial proliferation and hyperkeratosis are linked to gastric ulcers in pigs.

Hair follicle cells resist turning into skin cells.

Too much Smad7 changes skin and hair development by breaking down a protein called β-catenin, leading to more oil glands and fewer hair follicles.

MicroRNAs are important for skin health and could be targets for new skin disorder treatments.

Human skin has multiple layers and functions, with key roles in protection, temperature control, and appearance.

Mf-rich hair degrades more than Ma-rich hair, especially with Proteinase K.

The protein CTCF is essential for skin development, maintaining hair follicles, and preventing inflammation.

HSD11b1 affects skin nerves and increases non-histaminergic itch.

A virus protein can activate a pathway that may lead to abnormal hair follicle development.

Bioactive molecules show promise for improving skin repair and regeneration by overcoming current challenges with further research.

CD98hc's role in skin health decreases with age.

Cilostazol may help hair grow and could be a new treatment for hair loss.

Lgr5 is a marker for active, long-lasting stem cells in mouse hair follicles.

Certain microRNAs may protect against hair loss in alopecia areata and could be potential treatment targets.

Dermal Wnt/β-catenin signaling is important for the proper size and development of hair follicles.

Scientists can control how skin stem cells divide by using different treatments.

Hair follicle bulge cells don't help skin regrow after glucocorticoid damage; interfollicular epidermis cells do.

Bacteria can help skin regenerate through a process called IL-1β signaling.

Some viruses can cause cancer by changing cell processes and avoiding the immune system; vaccines and targeted treatments help reduce these cancers.

Using developmental signaling pathways could improve adult wound healing by mimicking scarless embryonic healing.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

Hypothyroidism harms rat skin, but topical triiodothyronine may help improve it.

Boosting HGF signaling could improve the creation of hair follicles in lab-made skin.

Activating TRPA1 reduces scarring and promotes tissue regeneration.

Pannexin 3 helps skin and hair growth by controlling a protein called Epiprofin.

A specific group of skin stem cells was found to help maintain hair follicle cells.

Overexpression of SSAT causes hair loss and skin issues, but reducing putrescine can help.