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- Discovery Potential Hub Genes and Pathways in Keloid Fibroblast Development Based on Bioinformatics Analysis
- 442 Deciphering the role of keloid fibroblasts in itch
- Single‐cell RNA sequencing highlights the role of proinflammatory fibroblasts, vascular endothelial cells, and immune cells in the keloid immune microenvironment
- A Signature of Genes Featuring FGF11 Revealed Aberrant Fibroblast Activation and Immune Infiltration Properties in Keloid Tissue
- Catecholaminergic neurons boost fibroblast osteogenic activity in keloid
- Single cell deciphering of pruritic keloids: the interaction between fibroblasts and Schwann cells through the Midkine signaling
- Gut microbiota-derived metabolites in keloid and hypertrophic scarring
- Extracellular vesicles derived from embryonic day 13 fetal mouse dermal fibroblasts exhibit potential antifibrotic activity
- Fibroblast Heterogeneity in Healthy and Wounded Skin
- Hypoxia macrophage-derived exosomal miR-26b-5p targeting PTEN promotes the development of keloids
- Exploring the Complexities of TGF-beta Signaling in Keloids: Beyond the Classical Smad Pathway
- LB1027 Identifying the mechanism of tension-associated tissue fibrosis based on single cell RNA sequencing on keloid scar
- Dissecting the Molecular Mechanisms Involved in Keloid Pathogenesis and Response to Therapy
- WNT5A drives interleukin-6-dependent epithelial–mesenchymal transition via the JAK/STAT pathway in keloid pathogenesis
- Fibroblasts become fat to reduce scarring
- Suppression of fibroblast proliferation and lysyl hydroxylase activity by minoxidil.
- Effects of Minoxidil on Cultured Human Skin Fibroblasts
- Pharmacotherapy for Keloids and Hypertrophic Scars
- MicroRNA-21 in Skin Fibrosis: Potential for Diagnosis and Treatment
- CD201+ fascia progenitors choreograph injury repair
- Toward understanding scarless skin wound healing and pathological scarring
- Regeneration of Dermis: Scarring and Cells Involved
- Pathological Skin Scarring: From Mechanism to Targeted Local Therapy
- Langerin+ Dendritic Cells in Cutaneous Fibrosis: The TGF-β1 Signaling Axis
- Minoxidil exerts different inhibitory effects on gene expression of lysyl hydroxylase 1, 2, and 3: Implications for collagen cross-linking and treatment of fibrosis
- Stem cell-derived exosomes: emerging therapeutic opportunities for wound healing
- Pathophysiology of the burn scar
- Krox20 expression in abnormal scars: An immunohistochemical study
- miR-3606-3p alleviates skin fibrosis by integratively suppressing the integrin/FAK, p-AKT/p-ERK, and TGF-β signaling cascades
- An introduction to skin disease in melanin rich skin