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Research 31–60 of 85
- Homozygous Dominant Missense Mutation in Keratin 17 Leads to Alopecia in Addition to Severe Pachyonychia Congenita
- Krt6a-Cre Transgenic Mice Direct LoxP-Mediated Recombination to the Companion Cell Layer of the Hair Follicle and Following Induction by Retinoic Acid to the Interfollicular Epidermis
- Building Models for Keratin Disorders
- KERATIN 17-related recessive atypical pachyonychia congenita with variable hair and tooth anomalies
- Keratins of the Human Hair Follicle
- The Catalog of Human Hair Keratins
- The expanding significance of keratin intermediate filaments in normal and diseased epithelia
- K6irs1, K6irs2, K6irs3, and K6irs4 Represent the Inner-Root-Sheath-Specific Type II Epithelial Keratins of the Human Hair Follicle1
- Autosomal-Dominant Woolly Hair Resulting from Disruption of Keratin 74 (KRT74), a Potential Determinant of Human Hair Texture
- TCF/Lef1 activity controls establishment of diverse stem and progenitor cell compartments in mouse epidermis
- Interleukin-1 Induces Transcription of Keratin K6 in Human Epidermal Keratinocytes
- Keratin 17 Expression in the Hard Epithelial Context of the Hair and Nail, and its Relevance for the Pachyonychia Congenita Phenotype
- A Novel Epithelial Keratin, hK6irs1, is Expressed Differentially in All Layers of the Inner Root Sheath, Including Specialized Huxley Cells (Flügelzellen) of the Human Hair Follicle
- A Missense Mutation within the Helix Initiation Motif of the Keratin K71 Gene Underlies Autosomal Dominant Woolly Hair/Hypotrichosis
- Characterization of a 300 kbp Region of Human DNA Containing the Type II Hair Keratin Gene Domain
- Understanding autoimmunity of vitiligo and alopecia areata
- Keratins and skin disease
- A novel control of human keratin expression: cannabinoid receptor 1-mediated signaling down-regulates the expression of keratins K6 and K16 in human keratinocytes<i>in vitro</i>and<i>in situ</i>
- Identification of Novel Mutations in Basic Hair Keratins hHb1 and hHb6 in Monilethrix: Implications for Protein Structure and Clinical Phenotype
- Type II Epithelial Keratin 6hf (K6hf) Is Expressed in the Companion Layer, Matrix, and Medulla in Anagen-Stage Hair Follicles
- Delayed Cutaneous Wound Healing and Aberrant Expression of Hair Follicle Stem Cell Markers in Mice Selectively Lacking Ctip2 in Epidermis
- Loss of epidermal Evi/Wls results in a phenotype resembling psoriasiform dermatitis
- Significance of stress keratin expression in normal and diseased epithelia
- A Novel Type II Cytokeratin, mK6irs, is Expressed in the Huxley and Henle Layers of the Mouse Inner Root Sheath
- An unexpected role for keratin 10 end domains in susceptibility to skin cancer
- Increased rate of hair keratin gene loss in the cetacean lineage
- Alopecia in a Novel Mouse Model RCO3 Is Caused by mK6irs1 Deficiency
- Keratins as an Inflammation Trigger Point in Epidermolysis Bullosa Simplex
- Normal Fur Development and Sebum Production Depends on Fatty Acid 2-Hydroxylase Expression in Sebaceous Glands
- Against the Rules: Human Keratin K80