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Hair follicle stem cells are key for hair and skin regeneration, can be reprogrammed, and have potential therapeutic uses, but also carry a risk of cancer.

Early development of hair, teeth, and glands involves specific signaling pathways and cellular interactions.

Telocytes might help with skin repair and regeneration.

Estrogens significantly influence hair growth by interacting with receptors in hair follicles and may help regulate the hair growth cycle.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

Some people with pattern hair loss may also have scalp inflammation and scarring similar to lichen planopilaris.

Memory T cells in the skin balance staying put and moving into the blood, clustering around hair follicles, and increasing in number after infection.

Nanoparticles improve drug delivery through the skin but more research is needed on their long-term effects and skin penetration challenges.

Stem cell self-renewal is complex and needs more research for full understanding.

The dermal papilla is crucial for hair growth and health, and understanding it could lead to new hair loss treatments.

Cathepsin L is essential for normal hair growth and development.

The Sonic hedgehog pathway is crucial for new hair growth during mouse skin healing.

Aging causes skin stem cells to work less effectively.

Memory regulatory T cells need IL-7, not IL-2, to stay in peripheral tissues.

The niche environment controls stem cell behavior and plasticity, which is important for tissue health and repair.

Sonic hedgehog signaling is crucial for hair growth and maintaining hair follicle identity.

PRP treatment improves hair growth, and the device used can affect results, with some being more effective.

Stem cells in the hair follicle are regulated by their surrounding environment, which is important for hair growth.

Modulating BMP activity changes the number, size, shape, and type of ectodermal organs.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

Damage to skin releases dsRNA, which activates TLR3 and helps in skin and hair follicle regeneration.

The skin's internal clock affects healing, cancer risk, aging, immunity, and hair growth, and disruptions can harm skin health.

Low doses of some substances can be beneficial, while high doses can be harmful or toxic.

Mice hair growth patterns get more complex with age and can change with events like pregnancy or injury.

Normal skin cell renewal doesn't need RAR signaling, but vitamin A-related skin thickening does.

Stress can cause hair loss by negatively affecting hair follicles and this effect might be reversed with specific treatments.

Fat cells in the skin help start healing and form important repair cells after injury.

Stem cell plasticity is crucial for wound healing but can also contribute to cancer development.

The study found that Frontal Fibrosing Alopecia affects a broader age range of women and early treatment can help stop hair loss.

The document describes how to tell different types of non-scarring hair loss apart by looking at hair and scalp tissue under a microscope.