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Uncombable hair syndrome causes frizzy hair and can affect the nervous system, eyes, and ears, often co-occurring with other hair, skin, nail, and teeth conditions, and is linked to three specific gene mutations.

Removing a specific gene in certain skin cells causes hair loss on the body by disrupting normal hair development.

Dermal Wnt/β-catenin signaling is important for the proper size and development of hair follicles.

Keratin proteins are crucial for hair structure and strength.

Beta-caryophyllene helps improve wound healing in mice, especially in females.

MMP-9 is essential for proper hair canal formation.

β-catenin in the dermal papilla is crucial for normal hair growth and repair.

Human hair keratins K85 and K35 create unique filament patterns important for early hair formation.

BLIMP1 is essential for skin maintenance but not for defining sebaceous gland progenitors.

GDNF helps grow hair and heal skin wounds by acting on hair stem cells.

Psychotropic drugs can cause hair loss or excessive hair growth.

External factors can cause skin cancer cells that usually don't spread to grow and form tumors in mice.

Genes related to calcium signaling and lipid metabolism are important for curly hair in Mangalitza pigs.

Mutations in three genes cause Uncombable Hair Syndrome, leading to frizzy hair that can't be combed flat.

Human hair follicle stem cells can be isolated using specific markers for potential therapeutic use.

Mice with too much Claudin-6 have skin barrier problems and abnormal hair growth.

Understanding hair follicle biology and stem cell control could lead to new hair loss treatments.

Scientists successfully grew new hair follicles in regenerated mouse skin using mouse and human cells.

Endo-HSE helps grow hair-like structures from human skin cells in the lab.

Fox genes are important for hair growth and development in cashmere goats.

AP-2α and AP-2β proteins are essential for healthy adult skin and hair.

Melatonin makes cashmere grow earlier and more by increasing certain gene activity in goats.

Overexpressing Merkel cell virus proteins in human hair follicles can create clusters of cells that resemble Merkel cell cancer.

Researchers found WNT10A to be a key gene in developing goat hair follicles.

Sox21 is crucial for tooth development and enamel formation by preventing cells from changing into a different type.

GPRC5D is linked to the formation of hair, nails, and certain tongue areas.

A specific chemical change in the S100A3 protein leads to the formation of a four-part structure important for hair formation.

Human hair matrix cells and dermal papilla fibroblasts can form early hair follicle structures but don't produce hair shafts yet.

Keratin-associated proteins are part of the developing hair fiber cuticle.

Mutations in specific keratin genes cause improper hair structure in mice due to faulty keratin protein assembly.