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The new triangular flag-shaped design for incisions in hair transplant surgery provides better hair alignment and cosmetic appearance without extra scarring, especially for patients with specific hair directions.

Exosomes are crucial for protecting sensory hair cells in the inner ear.

Gene therapy, especially using atoh1, shows promise for creating functional sensory hair cells in the inner ear, but dosing and side effects need to be managed for clinical application.

Blocking autophagy increases survival of inner ear hair cells exposed to gentamicin.

Increasing Rps14 helps grow more inner ear cells and repair hearing cells in baby mice.

PCS rats show significant inner ear damage and zinc deficiency, similar to liver cirrhosis patients.

Ptprq has multiple forms that change during inner ear development.

A low dose of rapamycin increases inner ear hair cell creation by boosting SOX2+ cell numbers.

CaBP1 and CaBP2 are necessary for proper hearing and neurotransmission in the ear's inner hair cells.

CaBP1 and CaBP2 are necessary for proper hearing and neurotransmission in the ear's inner hair cells.

CaBP1 and 2 are necessary for maintaining calcium currents and hearing in inner ear cells.

Lack of zinc can cause hearing loss by damaging important parts of inner ear cells in mice.

CaBP1 and CaBP2 are important for maintaining hearing by supporting continuous calcium currents and nerve signaling in the ear.

CaBP1 and 2 are important for maintaining the activity of calcium channels necessary for hearing in inner ear cells.

CaBP1 and CaBP2 are important for continuous hearing by preventing inactivation of calcium currents in ear cells, with CaBP2 also able to restore hearing when reintroduced.

Sesn2 protects inner ear hair cells from damage by regulating certain cell survival pathways.

Melatonin may protect inner ear cells from damage by reducing cell death and oxidative stress, potentially treating sudden hearing loss.

Deferoxamine may help protect inner ear cells from damage caused by oxidative stress.

Activin A increases inner ear hair cell development, while follistatin decreases it.

Zebrafish need MYC and FGF to regenerate inner ear hair cells.

Follistatin and LIN28B together improve the ability of inner ear cells in mice to regenerate into hearing cells.

Mutations in the PTPRQ gene cause significant balance issues in mice due to hair bundle defects in the inner ear.

Activin A promotes ear hair cell development, while follistatin delays it.

Activin A and follistatin control when ear hair cells form in mice.

Light can be used to stimulate ear hair cells, improving speed and consistency over previous methods.

Some natural compounds can protect fish ear cells from damage by certain antibiotics without affecting the antibiotics' ability to fight infections.

Mutations in the hairless gene in mice affect its expression and lead to a range of developmental issues in multiple tissues.

Certain cells in the adult mouse ear come from cranial neural crest cells, but muscle and hair cells do not.

Low level laser therapy may help regenerate hair cells in the ear after damage from gentamicin.

Melatonin may protect ear cells from damage caused by nicotine.