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Research 571–600 of 1000+
- Moniletrix of the scalp from almost normal aspect to total alopecia: variable intrafamilial expressiveness
- Fundamentals
- Increasing of PCOs among Young Females
- The role of functional variants in the aetiology of polycystic ovary syndrome
- ATRICHIA WITH PAPULAR LESIONS – A CASE REPORT
- In This Issue
- Case Report: 7 Year-Old Saudi Female with Monilethrix
- COMPARATIVE STUDY OF TROPONIN I LEVEL IN ALOPECIA AREATA, ANDROGENIC ALOPECIA AND HEALTHY CONTROL SUBJECTS
- Androgenetic alopecia: What impact does it have on the quality of Life?
- Female Androgenetic (?) Alopecia
- A genetic test for androgenetic alopecia: polymorphisms in the androgen receptor gene provide a genetic screening test for androgenetic alopecia and earlier medical intervention
- Androgenetic Alopecia (AGA)
- Biology of hair pigmentation and its role in premature canities
- Correction of Hair Shaft Defects through Allele-Specific Silencing of Mutant Krt75
- Exploring the Link Between Selenium from Wheat and Hair Loss
- Polymorphism of the Androgen Receptor Gene is Associated with Male Pattern Baldness
- Human Hair Growth Deficiency Is Linked to a Genetic Defect in the Phospholipase Gene <i>LIPH</i>
- Keratin gene mutations in disorders of human skin and its appendages
- Androgenetic alopecia: a review
- Cantú Syndrome Is Caused by Mutations in ABCC9
- Ichthyosis
- FGF5 is a crucial regulator of hair length in humans
- Evidence for Novel Functions of the Keratin Tail Emerging from a Mutation Causing Ichthyosis Hystrix
- RBPJ Mutations Identified in Two Families Affected by Adams-Oliver Syndrome
- Male-pattern baldness susceptibility locus at 20p11
- Androgenetic Alopecia: An Update of Treatment Options
- Uncombable hair syndrome: Observations on response to biotin and occurrence in siblings with ectodermal dysplasia
- Pulmonary manifestations of Birt-Hogg-Dubé syndrome
- Mutations in ABCB6 Cause Dyschromatosis Universalis Hereditaria
- Clinical and Immunological Phenotype of Patients With Primary Immunodeficiency Due to Damaging Mutations in NFKB2