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Research 751–780 of 1000+
- Eyes open on stem cells
- 59 Scarring alopecia is driven by the collapse of EGFR-protected JAK-STAT1-sensitive stem cell immune privilege
- Maintenance of Hair Follicle Immune Privilege Is Linked to Prevention of NK Cell Attack
- Immunology of the Human Nail Apparatus: The Nail Matrix Is a Site of Relative Immune Privilege
- Lichen planopilaris is characterized by immune privilege collapse of the hair follicle's epithelial stem cell niche
- Hair Follicle Immune Privilege Revisited: The Key to Alopecia Areata Management
- Hair Follicle Mesenchyme-Associated PD-L1 Regulates T-Cell Activation Induced Apoptosis: A Potential Mechanism of Immune Privilege
- Does collapse of immune privilege in the hair-follicle bulge play a role in the pathogenesis of primary cicatricial alopecia?
- Vasoactive intestinal peptide, whose receptor-mediated signalling may be defective in alopecia areata, provides protection from hair follicle immune privilege collapse
- Somatostatin Expression in Human Hair Follicles and Its Potential Role in Immune Privilege
- Regulatory T cells in skin mediate immune privilege of the hair follicle stem cell niche
- Can the Hair Follicle Become a Model for Studying Selected Aspects of Human Ocular Immune Privilege?
- A comparative analysis of immune privilege in pregnancy and cancer in the context of checkpoint blockade immunotherapy
- Exacerbation of alopecia areata during pegylated interferon alpha-2b and ribavirin therapy, possibly due to the collapse of hair follicle immune privilege
- Epidermal growth factor receptor/mitogen-activated kinase inhibitor treatment induces a distinct inflammatory hair follicle response that includes collapse of immune privilege
- Immune privilege and alopecia areata
- Microneedle‐mediated Delivery of Immunomodulators Restores Immune Privilege in Hair Follicles and Reverses Immune‐Mediated Alopecia
- Impact of epidermal growth factor receptor and mitogen-activated kinase inhibition on hair follicles (HFs): partial HF immune privilege collapse and excessive interleukin-33 secretion
- 90 Exploring the therapeutic potential of DHODH inhibitor farudodstat for alopecia areata treatment in a novel ex vivo model of human hair follicle immune privilege collapse
- LB1777 A novel ex vivo model of human hair follicle immune privilege collapse reveals the potential of farudodstat, a DHODH inhibitor, as a therapeutic for alopecia areata treatment
- 051 γδ T cells as novel players in alopecia areata pathobiology: Vδ1 + T lymphocytes may recognize “stressed” hair follicle keratinocytes, leading to IFNγ-dependent hair follicle dystrophy and immune privilege collapse
- Immune Regulation in Hair Follicle Protection and Hair Regeneration: Insights from the Collapse of Immune Privilege in Alopecia Areata and Targeted Immunotherapy
- 63418 JAK3/TEC kinase pathways are active in alopecia areata lesions, and their inhibition with Ritlecitinib prevents αCD3/αCD28+IL-2 induced immune privilege collapse in healthy hair follicles ex vivo
- Scarring hair follicle destruction is driven by the collapse of EGFR-protected JAK-STAT1-sensitive stem cell immune privilege
- ISID1338 - Epidermal growth factor receptor protects from immune privilege collapse and scarring hair follicle destruction
- ISIDLB1777 - A novel ex vivo model of human hair follicle immune privilege collapse reveals the potential of farudodstat, a DHODH inhibitor, as a therapeutic for alopecia areata treatment
- 1338 Epidermal growth factor receptor protects from immune privilege collapse and scarring hair follicle destruction
- 1419 Pre-clinical in-vitro assays for high throughput screening of drug candidates for protection and restoration of immune privilege in alopecia areata
- 546 Epidermal growth factor receptor as a key regulator of the immune privilege
- 013 IL-15/IL-15Rα signaling is a guardian of human hair follicle immune privilege and promotes hair growth