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The research updated the skin cell profile, finding new skin cell markers and showing fibroblasts' key role in skin health.

EGFR deficiency causes significant changes in skin cells and hair follicles.

T cells and bacteria in the gut and skin help maintain health and protect against disease.

Blocking IL-12 can help treat alopecia areata by preventing hair follicle immune issues.

Aged individuals heal wounds less effectively due to specific immune cell issues.

The research suggests immune system changes and specific gene expression may contribute to male hair loss, proposing potential new treatments.

Mesenchymal Stem/Stromal Cells (MSCs) help in wound healing and tissue regeneration, but can also contribute to tumor growth. They show promise in treating chronic wounds and certain burns, but their full healing mechanisms and potential challenges need further exploration.

The research identified unique metabolic activities in immune cells associated with hair loss in Alopecia Areata.

Cicatricial alopecia, a permanent hair loss condition, is mainly caused by damage to specific hair follicle stem cells and abnormal immune responses, with gene regulator PPAR-y and lipid metabolism disorders playing significant roles.

Dandruff is linked to changes in the immune system of hair follicles and skin.

New drug targets for hair loss treatment were identified by studying cell interactions in hair follicles.

Immune changes and specific genes contribute to male hair loss.

Immune activities and specific genes are important in male pattern baldness.

Skin stem cells interacting with their environment is crucial for maintaining and regenerating skin and hair, and understanding this can help develop new treatments for skin and hair disorders.

CRH in the skin acts like the body's stress response system, affecting cell behavior and immune activity.

Lichen Planopilaris and Frontal Fibrosing Alopecia may help us understand hair follicle stem cell disorders and suggest new treatments.

Vitamin D and its receptor are essential for hair growth, cell regulation, immune function, and heart health.

Human dermal γδT-cells respond to stress in hair follicles, contributing to hair loss.

Cholesterol-related compounds can stop hair growth and cause inflammation in a type of scarring hair loss.

Certain immune cells contribute to severe hair loss in chronic alopecia areata, with Th17 cells possibly having a bigger impact than cytotoxic T cells.

Mesenchymal stem cell therapy may help treat alopecia areata by promoting hair growth and reducing inflammation.

Newborns with the common rash Erythema Toxicum have many active mast cells in their skin, but these cells don't produce the LL-37 peptide.

Hairless HRS/J mice resist Bacillus anthracis skin infections due to high numbers of immune cells, not because they lack hair follicles.

Recent findings suggest that genetic factors, immune system issues, and skin cell defects might contribute to the development of hidradenitis suppurativa.

Skin stem cells remember past inflammation, helping them respond better to future injuries and possibly aiding in treating skin issues.

Deleting the BRD4 protein in certain skin cells causes hair loss and skin inflammation.

Regulatory γδ T cells help protect hair follicles from alopecia areata and promote hair regrowth.

Cellular and immunotherapies show promise for healing chronic wounds but need more research.

Different stem cells are key for hair growth and health, and understanding their regulation could help treat hair loss.

γδTregs can protect hair follicles from alopecia areata and may help regrow hair.