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Non-immune dermal cells dominate, epidermal cells increase after day 9, and certain immune cells persist beyond inflammation in wound-induced hair follicle regeneration.

A specific DNA sequence caused hair loss in male mice by activating immune cells and increasing a certain immune signal.

Hair follicles help attract immune cells to the skin during stress.

Certain immune cells in the skin release a protein that stops hair growth by keeping hair stem cells inactive.

An imbalance between certain immune cells is linked to a chronic skin condition and may be influenced by obesity, smoking, and autoimmune issues.

The study concluded that immune cells attacking hair follicles cause hair loss in alopecia, with genetics and environment also playing a role, and highlighted the potential of certain treatments.

Special immune cells called Regulatory T cells help control skin inflammation and repair in various skin diseases.

Imiquimod cream activates hair follicle stem cells and causes early hair growth by changing immune cells and certain protein expressions.

Hair loss in male pattern baldness is linked to changes in immune cell behavior around hair follicles.

Removing certain immune cells in mice causes their hair to enter the growth phase earlier than usual.

Skin cells and immune cells change in a skin condition called hidradenitis suppurativa, and a certain treatment can improve these changes.

IL-17 and certain immune cells are linked to more severe alopecia areata.

Higher levels of certain immune cells and proteins are linked to more severe lupus symptoms.

Some brain cancer cells avoid immune system detection, and certain treatments could target this to slow their growth; also, certain fat cell precursors help regenerate hair and skin after injury.

Corticosteroid therapy reduces specific immune cells and promotes hair growth in alopecia areata patients.

Jagged-1 in skin Tregs is crucial for timely wound healing by recruiting specific immune cells.

Scarred skin in lichen planopilaris loses immune cells due to a decrease in a specific protein in skin cells.

A protein from certain immune cells is key for new hair growth after skin injury in mice.

Patients with Alopecia areata have fewer specific immune cells that normally regulate the immune system, which may contribute to the condition.

Using CD123 to detect certain immune cells helps diagnose a type of hair loss condition.

Lack of Vitamin D receptor changes skin structure and increases certain immune cells in the skin.

Activating TLR3 improves the healing and immune properties of periodontal ligament stem cells.

Severe alopecia areata involves higher levels of certain immune cells, which can be normalized with betamethasone.

Higher PD-1 levels are linked to fewer immune cells in hair follicles in alopecia areata.

The study suggests that a specific type of immune cell, memory-like NK cells, may increase during active hair loss in Alopecia areata.

Gefitinib and Sasam-Kyeongokgo together significantly reduce cancer growth and improve immune response in mice.

A substance called FGF9 from certain immune cells can trigger new hair growth during wound healing in mice, but humans may not have the same response due to fewer of these cells.

Stromal stem cells may help heal wounds by becoming structural cells or affecting the immune system, but more research is needed to understand how.

Antroquinonol may help prevent skin depigmentation by suppressing certain immune cells.

Activating TLR9 helps heal wounds and regrow hair by using specific immune cells.