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Acne patients' skin, both with and without lesions, shows a strong immune response and higher antimicrobial activity.

Alopecia areata is an autoimmune condition causing hair loss, influenced by genetics, stress, and diet, and may be prevented by a high soy oil diet.

SH-MSCs gel can effectively treat alopecia by increasing IL-10 and decreasing TNF-α gene expression.

Blocking IL-17 can reduce skin inflammation in a mouse model of pityriasis rubra pilaris.

The role of γδT-cells in causing alopecia areata remains unclear.

Regulatory T cells are essential for normal and improved wound healing in mice.

Special immune cells called Treg cells are important for maintaining and regenerating hair by activating a specific growth signal in hair stem cells.

Androgens may influence T cells, contributing to higher autoimmune liver disease risk in women.

TNC+ fibroblasts play a key role in skin inflammation by interacting with T cells.

Thymic transplantation normalized some T-cells but not others, maintaining immune function.

Human dermal γδT-cells respond to stress in hair follicles, contributing to hair loss.

Bacteria can help skin regenerate through a process called IL-1β signaling.

IL-15 helps protect hair follicles from immune attacks and encourages hair growth.

Certain immune cells contribute to skin autoimmune diseases, and some treatments can reverse hair loss in these conditions.

Foxp3+ Regulatory T Cells are important for immunity and tolerance, affect hair growth and wound healing, and their dysfunction can contribute to obesity-related diseases and other health issues.

Imiquimod cream activates hair follicle stem cells and causes early hair growth by changing immune cells and certain protein expressions.

Inflammation plays a key role in activating skin stem cells for hair growth and wound healing, but more research is needed to understand how it directs cell behavior.

IL-33 is linked to hair follicle damage in psoriasis and could be a treatment target for hair loss in this condition.

Cutaneous lupus patients have higher levels of certain immune cells in their blood and skin.

Valproic Acid could potentially be used to treat immune-related conditions due to its ability to modify immune cell functions.

Changes to the Foxp3 protein affect how well regulatory T cells can control the immune system, which could help treat immune diseases and cancer.

Alopecia areata involves immune activation in the scalp, suggesting treatments targeting TH1, TH2, and IL-23 pathways.

IL-17C is important in inflammatory skin diseases and could be a target for treatment.

Skin bacteria, specifically Staphylococcus aureus, help in wound healing and hair growth by using IL-1β signaling. Using antibiotics on skin wounds can slow down this natural healing process.

Skin bacteria can help wound healing by activating certain immune cells.

Ifidancitinib, a JAK inhibitor, effectively regrows hair in mice with alopecia by tiring out harmful T cells.

CRAC channels are crucial for the development and function of specialized immune cells, preventing severe inflammation and autoimmune diseases.

Tissue environment greatly affects the unique epigenetic makeup of regulatory T cells, which could impact autoimmune disease treatment.

Jagged-1 in skin Tregs is crucial for timely wound healing by recruiting specific immune cells.

New topical treatment using spherical nucleic acids shows promise in reducing psoriasis inflammation.