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Alopecia areata involves complex immune and genetic factors, with potential treatment targets identified, but more research is needed.

Certain immune system proteins are important for skin healing but can cause problems if there are too many of them.

Understanding the immune-related causes of Alopecia Areata has led to potential treatments like JAK inhibitors.

Antroquinonol may help prevent skin depigmentation by suppressing certain immune cells.

Acne patients' skin, both with and without lesions, shows a strong immune response and higher antimicrobial activity.

Type 3 Innate Lymphoid Cells help maintain skin health and balance, and are involved in skin diseases and healing.

TSLP affects atopic dermatitis by increasing sebum and reducing fat through IL-4/IL-13 signaling.

Innate lymphoid cells help control skin bacteria by regulating sebaceous glands.

Alopecia areata involves both innate and adaptive immunity, with specific genes linked to the disease.

People with atopic dermatitis have more CD4+ T cells that respond to a certain bacterial lipid, which may play a role in the skin condition's inflammation.

Faulty Notch signalling may cause hair follicle changes and inflammation in hidradenitis suppurativa.

Treg cells help repair and regenerate tissues by interacting with local cells.

Interleukin levels are higher in alopecia areata patients but don't predict disease severity or duration.

Tofacitinib regrew hair in a man with total hair loss but raised cytokine levels, needing more research on possible side effects.

Alopecia areata is an autoimmune condition causing hair loss, influenced by genetics, stress, and diet, and may be prevented by a high soy oil diet.

Chronic inflammatory skin diseases are caused by disrupted interactions between skin cells and immune cells.

New topical treatment using spherical nucleic acids shows promise in reducing psoriasis inflammation.

Bacteria can help skin regenerate through a process called IL-1β signaling.

Skin bacteria can help wound healing by activating certain immune cells.

People with atopic dermatitis have different skin bacteria, and targeting these bacteria might help treat the condition.

Human dermal γδT-cells respond to stress in hair follicles, contributing to hair loss.

IL-15 helps protect hair follicles from immune attacks and encourages hair growth.

Ifidancitinib, a JAK inhibitor, effectively regrows hair in mice with alopecia by tiring out harmful T cells.

Ginseng, especially its component ginsenosides, can promote hair growth, reduce hair loss, and potentially treat conditions like alopecia by affecting cell pathways and cytokines.

Regulatory T cells protect hair follicle stem cells by maintaining immune privilege in the skin.

Certain immune cells contribute to skin autoimmune diseases, and some treatments can reverse hair loss in these conditions.

SH-MSCs gel can effectively treat alopecia by increasing IL-10 and decreasing TNF-α gene expression.

Retinol may affect immune responses in people with frequent skin boils.

Lichen planus is a chronic autoimmune disease that is hard to treat and more common in women.

Blocking IL-17 can reduce skin inflammation in a mouse model of pityriasis rubra pilaris.