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Research 31–60 of 1000+
- Tumour Necrosis Factor Alpha, Interferon Gamma and Substance P Are Novel Modulators of Extrapituitary Prolactin Expression in Human Skin
- Translational Positioning of Janus Kinase (JAK) Inhibitors in Alopecia Areata
- Inheritance-Specific Dysregulation of Th1- and Th17-Associated Cytokines in Alopecia Areata
- Concomitant Vitiligo and Psoriasis in a Patient Treated with Interferon Alfa‐2a for Chronic Hepatitis B Infection
- Treatment of Alopecia Areata with Anti-Interferon-γ Antibodies
- CGF therapy: bridging androgenetic alopecia observations to psoriasis treatment via IL-17 pathway
- Evaluation of the Relationship between Alopecia Areata and Viral Antigen Exposure
- Alopecia universalis after discontinuation of pegylated interferon and ribavirin combination therapy for hepatitis C: a case report
- Coexistence of dermatomyositis and alopecia areata: Insight into pathogenesis
- Trained Immunity and Local Innate Immune Memory in the Lung
- SnapshotDx Quiz: February 2018
- Biological therapy in the treatment of melanoma
- Hepatology highlights
- Serum levels of ifn-γ, complement component c3 c4 and vitamin d3 in the prognosis of patients with alopecia areata prospective teachers
- Mycophenolate Antagonizes IFN-γ-Induced Catagen-Like Changes via β-Catenin Activation in Human Dermal Papilla Cells and Hair Follicles
- Extracellular vesicles from IFN-γ-primed mesenchymal stem cells repress atopic dermatitis in mice
- Human Placenta Extract (HPH) Suppresses Inflammatory Responses in TNF-α/IFN-γ-Stimulated HaCaT Cells and a DNCB Atopic Dermatitis (AD)-Like Mouse Model
- 068 Pathogenic Th17 cells, CD8+CD69+CD49a- tissue-resident memory T cells and common γ chain receptor + natural killer cells express more IL-17, compared to IFN-γ, under the foxp3+ memory regulatory T cells-depleted microenvironment in patients with chronic alopecia areata
- Effects of Topical application of Astragalus membranaceus in Spontaneous Alopecia Mice Model
- Mesenchymal Stem Cells Antagonize IFN-Induced Proinflammatory Changes and Growth Inhibition Effects via Wnt/β-Catenin and JAK/STAT Pathway in Human Outer Root Sheath Cells and Hair Follicles
- Predominant expression of interleukin (IL)‐17 in chronic alopecia areata compared to IFN‐γ in pathogenic Th17 cells, tissue‐resident memory T cells and natural killer cells
- 051 γδ T cells as novel players in alopecia areata pathobiology: Vδ1 + T lymphocytes may recognize “stressed” hair follicle keratinocytes, leading to IFNγ-dependent hair follicle dystrophy and immune privilege collapse
- 083 More Expression of IL-17 than IFN-γ from CD49a-negative Tissue-Resident Memory T (Trm) cells at the Hair Bulge and Hair Bulb Region in the Scalp of Chronic Alopecia Areata Patients according to the Foxp3+mTregs depletion
- Baricitinib Attenuates IFN-γ and Polyinosinic:polycytidylic Acid‒Induced Mitochondrial Damage and Inflammasome Activation in Human Keratinocytes
- 0085 Th1/Th2 balance, IFN-γ–MHC-I axis, and regulatory T cells define therapeutic response endotypes in alopecia areata
- Keratin 17 in disease pathogenesis: from cancer to dermatoses
- Oxidative DNA Damage Accelerates Skin Inflammation in Pristane-Induced Lupus Model
- Somatostatin Expression in Human Hair Follicles and Its Potential Role in Immune Privilege
- Increased blood levels of NKG2D+CD4+ T cells in patients with alopecia areata
- Decreased CD19+CD24hiCD38hi Regulatory B Cells in Alopecia Areata