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Hair follicles may help teach the immune system to tolerate new self-antigens, but this can sometimes cause hair loss.

Lotion with fucoidan from brown seaweed improved skin and reduced allergy symptoms in mice with dermatitis.

Chemotherapy can cause various skin problems, and recognizing them helps improve patient care.

Current and future treatments for alopecia areata focus on immunosuppression, immunomodulation, and protecting hair follicles.

Certain immune cells contribute to severe hair loss in chronic alopecia areata, with Th17 cells possibly having a bigger impact than cytotoxic T cells.

Wnt/beta-catenin signaling in the skin helps fat cell development during hair growth and repair.

Targeting the protein Caveolin-1 might help treat a type of scarring hair loss called Frontal Fibrosing Alopecia.

A specific DNA sequence caused hair loss in male mice by activating immune cells and increasing a certain immune signal.

Low-dose naltrexone might be a cheap and effective additional treatment for hair loss with scalp discomfort due to its anti-inflammatory effects and few side effects.

Activating TLR3 improves the healing and immune properties of periodontal ligament stem cells.

Alopecia areata severity and treatment response are linked to specific cytokine levels.

Mesenchymal stem cells may help treat hair loss by improving hair cell growth and reducing inflammation.

Lichen Planopilaris and Frontal Fibrosing Alopecia may help us understand hair follicle stem cell disorders and suggest new treatments.

Hair follicles are normally protected from the immune system, but when this protection fails, it can cause hair loss in alopecia areata.

Certain gene variations may increase the risk and severity of alopecia areata.

Trichloroethylene causes skin inflammation in mice by increasing certain immune proteins.

Alopecia areata shows a unique type 1 interferon signature, suggesting potential treatment by targeting this pathway.

Th22 cells are essential for Tβ15-induced hair growth in mice.

EGCG may help treat alopecia areata by blocking certain immune responses and reducing specific harmful immune cells.

Both vitiligo and alopecia areata involve an immune response triggered by stress and specific genes, with treatments targeting this pathway showing potential.

Bone marrow-derived stem cells improved healing and reduced scarring in second-degree burns in rats.

Alopecia areata and vitiligo share immune system dysfunction but differ in specific immune responses and affected areas.

NK cells play a role in skin diseases like eczema and psoriasis.

Mushroom beta-glucans from Ganoderma lucidum can reduce tumor size and metastasis in cancer.

Certain immune-related proteins are higher in people with alopecia and their healthy relatives, hinting at a genetic link.

New methods to improve the healing abilities of mesenchymal stem cells for disease treatment are promising but need more research.

The new gel combining calcipotriene and betamethasone is effective and safe for treating scalp psoriasis.

Calcipotriol plus betamethasone with microneedling works better than tacrolimus for treating vitiligo.

Microneedling with betamethasone led to almost complete hair regrowth in severe alopecia areata.

TNFα, IFNγ, and Substance P significantly affect prolactin levels in human skin, suggesting new treatments for skin and hair conditions.