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Research 781–810 of 943
- EGFR Controls Hair Shaft Differentiation in a p53-Independent Manner
- Home-based devices in dermatology: a systematic review of safety and efficacy
- Squamous epithelial proliferation induced by walleye dermal sarcoma retrovirus cyclin in transgenic mice
- Molecular Mechanisms Regulating Hair Follicle Development
- Sox9 regulates melanocytic fate decision of adult hair follicle stem cells
- Madarosis and facial keratinaceous spines
- Tracing the cellular origin of cancer
- Fibroblasts and wound healing: an update
- Androgens modulate keratinocyte differentiation indirectly through enhancing growth factor production from dermal fibroblasts
- Lineage potential, plasticity and environmental reprogramming of epithelial stem/progenitor cells
- Keratin 17 modulates hair follicle cycling in a TN<i>Fα</i>-dependent fashion
- Thehairless gene of the mouse: Relationship of phenotypic effects with expression profile and genotype
- Spiny Keratoderma
- Effects of Acute UVB on Retinoid Metabolism
- Human stratified squamous epithelia differ in cellular fatty acid composition
- sm“FISH”ing for Hedgehog
- Biochemical and immunohistochemical analyses of keratin expression in basal cell carcinoma
- Life before and beyond blistering: The role of collagen XVII in epidermal physiology
- Altered Dermal Fibroblasts in Systemic Sclerosis Display Podoplanin and CD90
- Reciprocal role of vitamin D receptor on β-catenin regulated keratinocyte proliferation and differentiation
- The stem cell niche
- The Role of PPARβ/δ-Related Lipid Metabolism in High-Altitude Adaptation of Yak Coat Based on Proteomics and Metabolomics
- An Extended Epidermal Response Heals Cutaneous Wounds in the Absence of a Hair Follicle Stem Cell Contribution
- Activator Protein-1 Activity Regulates Epithelial Tumor Cell Identity
- Modulation of the differentiated phenotype of keratinocytes of the hair follicle and from epidermis
- Stratum-Specific Expression of Human Transferrin Receptor Increases Iron in Mouse Epidermis
- Inflammatory memory-activated biomimetic nanovesicles regulate neutrophil plasticity and metabolic reprogramming for rapid diabetic wound healing via targeting miR-193a-5p/TLR4/JNK/P38 MAPK pathways
- Protein Kinase C as a Therapeutic Target
- Targeting Wnt/β-catenin signaling and its interplay with TGF-β and Notch signaling pathways for the treatment of chronic wounds
- Wound healing with topical BRAF inhibitor therapy in a diabetic model suggests tissue regenerative effects