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Certain genetic variants are linked to frontal fibrosing alopecia in Spanish patients.

Frontal Fibrosing Alopecia is a hair loss condition mainly affecting postmenopausal women, with unclear causes and various clinical patterns.

The cause of Frontal fibrosing alopecia, a type of hair loss, is complex, likely involving immune responses and genetics, but is not fully understood.

A genetic marker linked to a type of hair loss was found in most patients studied.

Portraits show women's hairlines have moved forward over time, suggesting changes in the occurrence of frontal hair loss.

Frontal Fibrosing Alopecia's cause is unclear, affects mainly postmenopausal women, and current treatments focus on stopping hair loss rather than regrowth.

Genetic testing can help diagnose skin conditions but needs more research for full effectiveness.

The study concluded that immune cells attacking hair follicles cause hair loss in alopecia, with genetics and environment also playing a role, and highlighted the potential of certain treatments.

Oral contraceptive use may increase the risk of frontal fibrosing alopecia in women with a specific CYP1B1 gene variant.

Researchers found a specific immune receptor in patients that causes severe skin reactions to a drug.

B2m-free HLA variants may be a new class of HLA important in immune responses and diseases.

A protein called EGFR protects hair follicle stem cells, and when it's disrupted, hair follicles can be damaged, but blocking certain pathways can restore hair growth.

Trichoscopy helps tell apart Frontal Fibrosing Alopecia and Lichen planopilaris by showing different hair and scalp features.

Protein profiling of forehead skin can help distinguish between frontal fibrosing alopecia and androgenetic alopecia.

New understanding and treatments for hair loss are improving, but more research is needed.

Some drugs can cause rare but potentially deadly skin reactions, and early treatment and avoiding the drug again are key.

A gene variant causes patched hair loss in mice, similar to alopecia areata in humans.

A severe medication reaction required long treatment and led to hair loss and thyroid issues.

Hashimoto thyroiditis is an autoimmune disease that can lead to an underactive thyroid and is treated with medication and sometimes diet changes or surgery.

Certain genetic variants linked to immune response increase the risk of alopecia areata in Taiwanese people.

Certain immune system genes are linked to a higher risk of psoriasis and psoriatic arthritis, while others may offer protection.

Psychotropic medications can cause skin reactions, including severe conditions like SJS and TEN, and it's important for psychiatrists to recognize and manage these side effects.

COVID-19 vaccination does not significantly increase the risk of developing alopecia areata.

Stress can worsen skin conditions and affect mental health, so doctors should include stress management in skin treatment.

Antibiotics often cause skin reactions, making them a major health concern.

A gene variant increases the risk of a type of hair loss by affecting hair protein production.

Alopecia areata and vitiligo share immune system dysfunction but differ in specific immune responses and affected areas.

Current treatments for Alopecia Areata have mixed success, and there's a need for better, more accessible options and support for affected individuals.

Alopecia areata is an autoimmune disease where T cells attack hair follicles.

Mercury allergy linked to specific genes may contribute to burning mouth syndrome, and silicon might play a role in maintaining healthy hair.