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Research 211–240 of 1000+
- Enhancement of In Vitro Hair Shaft Elongation in Follicles Stored in Buffers That Prevent Follicle Cell Apoptosis
- Synchronizing actin and microtubules for axonal branching
- Topical Valproate Solution for Hair Growth
- Reporting Live from the Epidermal Stem Cell Compartment!
- Stem Cells in the Hair Follicle and Interfollicular Epidermis of Mice following Topical Application of Fluocinolone Acetonide
- Generalised Glucocorticoid Resistance
- The role of lipids in promoting hair growth through HIF-1 signaling pathway
- 250 Sweat glands shrink upward with aging, causing dermal layer defects that lead to loss of skin elasticity and wrinkling-establishment of 3D imaging method for skin appendages
- 254 WITHDRAWN
- Montagna Symposium 2015: Harnessing Stem Cells to Reveal Novel Skin Biology and Disease Treatments
- BJD Editor's Choice
- Enhancement of In Vitro Hair Shaft Elongation in Follicles Stored in Buffers That Prevent Follicle Cell Apoptosis
- The human keratins: biology and pathology
- Epidermal homeostasis: a balancing act of stem cells in the skin
- Scratching the surface of skin development
- Tcf3 and Lef1 regulate lineage differentiation of multipotent stem cells in skin
- Dedifferentiation of committed epithelial cells into stem cells in vivo
- Modulating the stem cell niche for tissue regeneration
- Molecular Dissection of Mesenchymal–Epithelial Interactions in the Hair Follicle
- Skin melanocytes: biology and development
- Stem Cells and the Niche: A Dynamic Duo
- Fibroblast heterogeneity: implications for human disease
- Aging and Immortality: Quasi-Programmed Senescence and Its Pharmacologic Inhibition
- Alopecia areata
- Wnt Signaling in Skin Development, Homeostasis, and Disease
- A family business: stem cell progeny join the niche to regulate homeostasis
- Transforming growth factor-β in stem cells and tissue homeostasis
- Tracing the cellular origin of cancer
- Pathobiology of chemotherapy-induced hair loss
- Particulate matter 2.5 damages skin cells by inducing oxidative stress, subcellular organelle dysfunction, and apoptosis