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Research 61–90 of 1000+
- Vitamin D resistance
- Evidence for 1,25-Dihydroxyvitamin D3-independent Transactivation by the Vitamin D Receptor
- The vitamin D receptor functions as a transcription regulator in the absence of 1,25-dihydroxyvitamin D3
- Lymphoid Enhancer-binding Factor-1 (LEF1) Interacts with the DNA-binding Domain of the Vitamin D Receptor
- Extraskeletal actions of vitamin D
- Use of Vitamins and Minerals in the Treatment of Hair Loss: A Cross-Sectional Survey among Dermatologists in Saudi Arabia
- Phenotypic Heterogeneity in 5 Japanese Patients with an Identical Point Mutation in the Vitamin D Receptor Gene
- Generation of Genetically Modified Rats Using CRISPR/Cas9 Genome-Editing System to Reveal Novel Vitamin D Actions
- Mineral Deficiencies: A Root Cause for Reduced Longevity in Mammals
- A human vitamin D receptor mutation causes rickets and impaired Th1/Th17 responses
- Mutations in the vitamin D receptor and hereditary vitamin D-resistant rickets
- Gene therapy for alopecia in type II rickets model rats using vitamin D receptor-expressing adenovirus vector
- Atrichia Caused by Mutations in the Vitamin D Receptor Gene is a Phenocopy of Generalized Atrichia Caused by Mutations in the Hairless Gene
- A Systemic Review on Topical Marketed Formulations, Natural Products, and Oral Supplements to Prevent Androgenic Alopecia: A Review
- Study of the Thyroid Profile of Patients with Alopecia
- A unique insertion/duplication in the VDR gene that truncates the VDR causing hereditary 1,25-dihydroxyvitamin D-resistant rickets without alopecia
- Hair follicles and their role in skin health
- Hereditary vitamin D resistant rickets (HVDRR) case series: phenotype, genotype, conventional treatment, and adjunctive cinacalcet therapy
- Diffuse loss of hair with papules
- Novel Vitamin D Receptor Mutations in Hereditary Vitamin D Resistant Rickets in Chinese
- The vitamin D receptor, the skin and stem cells
- The role of vitamin D receptor signaling in hair follicle health and alopecia: Current understanding and therapeutic implications
- Physiological Insights from the Vitamin D Receptor Knockout Mouse
- Vitamin D-dependent rickets type II. Defective induction of 25-hydroxyvitamin D3-24-hydroxylase by 1,25-dihydroxyvitamin D3 in cultured skin fibroblasts.
- Vitamin D metabolism and function in the skin
- Functional analyses of a novel missense and other mutations of the vitamin D receptor in association with alopecia
- Vitamin D receptor is essential for normal keratinocyte stem cell function
- Hereditary 1,25-dihydroxyvitamin D-resistant rickets with alopecia in four Egyptian families: report of three novel mutations in the vitamin D receptor gene
- Association of vitamin D level with alopecia areata
- Alopecia areata – Current understanding and management