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- Analysis of hairless corepressor mutants to characterize molecular cooperation with the vitamin D receptor in promoting the mammalian hair cycle
- The <i>hairless</i> gene mutated in congenital hair loss disorders encodes a novel nuclear receptor corepressor
- Physical and Functional Interaction between the Vitamin D Receptor and Hairless Corepressor, Two Proteins Required for Hair Cycling
- Interactions of the Vitamin D Receptor with the Corepressor Hairless
- Hairless is a nuclear receptor corepressor essential for skin function
- Modulation of Vitamin D Receptor Activity by the Corepressor Hairless: Differential Effects of Hairless Isoforms
- Hairless: A nuclear receptor corepressor essential for skin function
- Molecular Basis for Hair Loss in Mice Carrying a Novel Nonsense Mutation (<i>Hr<sup>rh-R</sup></i>) in the Hairless Gene (<i>Hr</i>)
- A Homozygous Nonsense Mutation in the Zinc-Finger Domain of the Human Hairless Gene Underlies Congenital Atrichia
- A novel missense mutation in the mouse hairless gene causes irreversible hair loss: Genetic and molecular analyses of Hrm1Enu
- Hairless Suppresses Vitamin D Receptor Transactivation in Human Keratinocytes
- Hairless and Wnt Signaling: Allies in Epithelial Stem Cell Differentiation
- Hairless Modulates Ligand-Dependent Activation of the Vitamin D Receptor-Retinoid X Receptor Heterodimer
- Mechanism of JmjC-containing protein Hairless in the regulation of vitamin D receptor function
- Hairless protein of Jumonji family and hair loss
- Quantification of the Vitamin D Receptor−Coregulator Interaction
- Ligand-Independent Actions of the Vitamin D Receptor Maintain Hair Follicle Homeostasis
- [Molecular cloning of full-long cDNA sequences encoding hairless gene in the Kunming mouse].
- Hairless triggers reactivation of hair growth by promoting Wnt signaling
- A unique insertion/duplication in the VDR gene that truncates the VDR causing hereditary 1,25-dihydroxyvitamin D-resistant rickets without alopecia
- Vitamin D receptor: molecular signaling and actions of nutritional ligands in disease prevention
- The role of vitamin D receptor mutations in the development of alopecia
- Vitamin D receptor-mediated control of Soggy, Wise, and Hairless gene expression in keratinocytes
- Hairless-knockout piglets generated using the clustered regularly interspaced short palindromic repeat/CRISPR-associated-9 exhibit abnormalities in the skin and thymus
- Disease causing homozygous variants in the human hairless gene
- Atrichia with papular lesions resulting from a novel insertion mutation in the human hairless gene
- Ligand‐independent Regulation of the hairless Promoter by Vitamin D Receptor<sup>†</sup>
- Taking advantage from phenotype variability in a local animal genetic resource: identification of genomic regions associated with the hairless phenotype in Casertana pigs
- Hairless controls hair fate decision via Wnt/β–catenin signaling
- Hairless and the polyamine putrescine form a negative regulatory loop in the epidermis