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Spiny mice are better at regenerating hair after injury than laboratory mice and could help us understand how to improve human skin repair.

MED1 affects skin wound healing differently in young and old mice.

Smad1 and Smad5 are essential for hair follicle development and stem cell sleepiness.

A protein called sFRP4 from skin cells stops the development of pigment-producing cells in hair.

New method efficiently isolates hair growth cells from newborn mouse skin.

Certain inhibitors applied to the skin can promote hair growth by maintaining a key hair growth signal.

Hair grows faster in the morning and is more vulnerable to damage from radiation due to the internal clock in hair follicle cells.

Injecting a mix of human skin and hair cells into mice can grow new hair.

A peptide called DPS-1 helps human scalp cells grow and stimulates hair growth in mice.

Cells in hair follicles help create fat cells in the skin by releasing a protein called Sonic Hedgehog.

The conclusion is that hair growth can be improved by activating hair cycles, changing the surrounding environment, healing wounds to create new hair follicles, and using stem cell technology.

Epidermal stem cells have potential for personalized regenerative medicine but need careful handling to avoid cancer.

A specific group of slow-growing stem cells marked by Thy1 is crucial for skin maintenance and healing in mice.

Stress hormone corticosterone blocks a growth factor to slow down hair stem cell activity and hair growth.

Lymphatic vessels are essential for hair follicle growth and skin regeneration.

Using defensins to activate stem cells may improve skin aging signs without causing inflammation.

Periodontal ligament stem cells show promise for regrowing tissues but require more research for safe, effective use.

3-Deoxysappanchalcone helps human hair cells grow and stimulates hair growth in mice by affecting certain cell signaling pathways.

DNA methylation is essential for skin and hair follicle development, and could be a target for treating skin diseases.

The review concluded that keeping the hair-growing ability of human dermal papilla cells is key for hair development and growth.

Disrupted stem cell signals in hairpoor mice cause hair loss.

More dermal papilla cells in hair follicles lead to larger, healthier hair, while fewer cells cause hair thinning and loss.

New treatments targeting skin stem cells show promise for skin repair, anti-aging, and cancer therapy.

Hair thinning causes stem cell loss through a process involving Piezo1, calcium, and TNF-α.

Boosting β-catenin signaling in certain skin cells can enhance hair growth.

Blocking β-catenin in skin cells improves hair growth during wound healing.

Prominin-1 expressing cells in the dermal papilla help regulate hair follicle size and communication but don't aid in skin repair.

Immune cells affect hair growth and could lead to new hair loss treatments.

Endoglin is crucial for proper hair growth cycles and stem cell activation in mice.

Enzymes involved in Vitamin A metabolism affect hair growth and type in mice.