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Vitamin D receptor is crucial for starting hair growth after birth.

miR-218-5p helps skin and hair growth by targeting SFRP2 and activating a specific signaling pathway.

Disruptions in hair follicle fibroblast dynamics can cause hair growth problems.

Wnt10b makes hair follicles bigger, but DKK1 can reverse this effect.

Mice without Vitamin D receptors have hair growth problems because of issues in the hedgehog signaling pathway.

Prolactin slows down hair growth in mice.

miR-22, a type of microRNA, controls hair growth and its overproduction can cause hair loss, while its absence can speed up hair growth.

Brg1 is crucial for hair growth and skin repair by maintaining stem cells and promoting regeneration.

Maintaining the right amount of retinoic acid is crucial for healthy hair and skin.

The study found that bioengineered hair follicles work when using cells from the same species but have issues when combining human and mouse cells.

Hair loss is common in lupus patients and can be permanent or reversible, depending on the type, with various treatments available.

Scientists found cells in hair that are key for growth and color.

Mutations in the PTPRQ gene cause significant balance issues in mice due to hair bundle defects in the inner ear.

Genetic variations affecting skin structure play a key role in severe acne.

Hoxc genes control hair growth through Wnt signaling.

Genetic mutations can cause hair growth disorders by affecting key genes and signaling pathways.

Heparan sulfate is important for hair growth, preventing new hair formation in mature skin, and controlling oil gland development.

Hair follicle regeneration in skin grafts may be possible using stem cells and tissue engineering.

Aging in hair follicle stem cells leads to hair graying, thinning, and loss.

Accurate clinical, histological, and genetic methods are key for understanding and treating hair disorders.

PDGF signaling is crucial for maintaining and renewing hair follicle stem cells, which could help treat hair loss.

Skin and hair can regenerate after injury due to changes in gene activity, with potential links to how cancer spreads. Future research should focus on how new hair follicles form and the processes that trigger their creation.

Prolactin may affect hair growth differently based on gender and scalp area.

Mice can correct hair follicle orientation without certain genes, but proper overall alignment needs those genes.

Mouse amnion can turn into skin and hair follicles with help from certain cells and factors.

Hormone therapy affects hair growth in transgender individuals, with testosterone potentially causing hair loss in trans men and estrogen reducing facial/body hair in trans women; treatment options vary.

Mice without collagen VI have slower hair growth normally but faster regrowth after injury.

The arrector pili muscle might play a role in hair loss and needs more research to understand its impact.

Steroidogenic isoenzymes may help improve treatments for common hair loss.

Both immature and mature fat cells are important for hair growth cycles, with immature cells promoting growth and mature cells possibly inhibiting it.