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Researchers found that hair follicle stem cells can become squamous cell carcinoma due to Ras activation, which could lead to new treatments.

Sebaceous glands play a key role in skin health, immunity, and various skin diseases.

People with pemphigus vulgaris and pemphigus foliaceus often have smaller sebaceous glands on their scalp.

Activating Nrf2 in skin cells causes skin disease similar to chloracne in mice.

The SHJH hr mice with a mutated Hr gene show signs of faster skin aging due to poor antioxidative protection.

Genes linked to wool fineness in sheep have been identified.

Marine-derived ingredients show potential for hair health but need more human trials to confirm effectiveness.

The conclusion is that exogen is a unique hair cycle phase and the new sampling method specifically targets this stage, which may help in future hair loss research.

Hair loss in autoimmune blistering skin diseases varies and may regrow with disease control.

t-Flavanone helps improve male pattern hair loss by making hair roots stronger.

A specific gene change plus an additional mutation in the same gene cause hereditary trichilemmal cysts.

Finasteride and minoxidil work best together for hair loss.

Scalp involvement is common in pemphigus and can lead to hair loss, with the severity of scalp lesions linked to overall disease severity.

The link between pemphigus and the patient's scarring hair loss is still unclear.

Mice with extra human KLK14 had hair and skin problems, including weaker cell bonds and inflammation, linked to Netherton syndrome.

Trichoscopy helps tell apart scalp lesions in pemphigus vulgaris and pemphigus foliaceus and is useful for choosing biopsy locations.

Scalp involvement in pemphigus means the disease is more severe and harder to treat.

Acne is significantly influenced by genetics, and understanding its genetic basis could lead to better, targeted treatments.

Different skin diseases show unique patterns of skin cell separation, cell death, and granular layer changes.

Hidradenitis suppurativa might be a type of autoinflammatory skin disease.

S100A3 protein is crucial for hair shaft formation in mice.

Cystatin M/E helps in the final stages of hair and nail formation by controlling certain enzymes.

Overactive Wnt signaling in mouse skin stem cells causes acne-like cysts and shrinking oil glands, which some treatments can partially fix.

Hair growth is controlled by specific gene clusters and proteins, and cysteine affects hair gene expression in sheep.

Abnormal Wnt signaling in hair follicle stem cells can lead to acne-like cysts.

Androgens increase a growth factor in hair cells by creating reactive oxygen species, and antioxidants might help treat hair loss.

Vitamin D Receptor is crucial for normal skin and hair growth.

Recognizing both trichostasis spinulosa and eruptive vellus hair cysts together is crucial for accurate diagnosis and treatment.

A specific type of skin cell creates an opening for hair to grow out, and problems with this process can lead to skin conditions.

Vitamin D Receptor is crucial for hair follicle shrinkage and cell death, affecting hair growth.