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Lgr5 is a marker for active, long-lasting stem cells in mouse hair follicles.

Mice can grow new hair follicles after skin wounds through a process not involving existing hair stem cells, but requiring more research to understand fully.

Ca_v1.2 affects hair growth and could be a target for hair loss treatments.

Integrin alphavbeta6 is important for wound healing and hair growth, and blocking it may improve these processes.

Disrupting Smad4 in mouse skin causes early hair follicle stem cell activity that leads to their eventual depletion.

The document concludes that hair follicles have a complex environment and our understanding of it is growing, but there are limitations when applying animal study findings to humans.

MicroRNA-148a is crucial for maintaining healthy skin and hair growth by affecting stem cell functions.

Skin and hair renewal is maintained by both fast and slow cycling stem cells, with hair regrowth primarily driven by specific stem cells in the hair follicle bulge. These cells can also help heal wounds and potentially treat hair loss.

Dlx3 is essential for hair growth and regeneration.

Hair follicle biology advancements may lead to better hair growth disorder treatments.

Notch signaling is essential for healthy skin and hair follicle maintenance.

Sebaceous glands can help harvest hair follicle stem cells to regenerate skin and hair.

The document concludes that the immune-inhibitory environment of the hair follicle may prevent melanoma development.

SCD1 is important for hair growth by keeping the connection in skin cells where hair stem cells live stable.

Hair loss in Androgenetic Alopecia is caused by genetics, aging, and lifestyle, leading to hair follicle shrinkage and related health risks.

Gamma rays did not change hair follicle density but increased white and hypopigmented hairs in mice.

Scientists created a long-lasting stem cell line from human hair that can turn into different skin and hair cell types.

Damaged hair follicle stem cells can cause permanent hair loss, but understanding their role could lead to new treatments.

A protein complex called mTORC1 likely affects when hair growth starts in mice.

HPV16 oncogenes disrupt the normal activity of hair follicle stem cells.

Merkel cells may have roles in sensing magnetic fields, creating fingerprints, Reiki energy healing, passing on environmental information to offspring, and influencing hair shape.

Blocking TSLP reduces skin inflammation and cell overgrowth in psoriasis.

Sebaceous glands can heal and regenerate after injury using their own stem cells and help from hair follicle cells.

Desmoglein 3 is important for keeping hair follicle stem cells inactive and maintaining their special properties.

N-acetyl-GED may help prevent and partially reverse a process that leads to scarring hair loss.

Increased androgen receptor expression in certain hair follicle areas may cause male pattern baldness.

PRP is a promising treatment for autoimmune hair loss but its exact workings are not fully understood.

Androgenetic alopecia causes hair loss by shrinking hair follicles due to androgens, with the connection between the muscle and hair follicle determining if the loss is reversible.

Different types of stem cells in hair follicles play unique roles in wound healing and hair growth, with some stem cells not originating from existing hair follicles but from non-hair follicle cells. WNT signaling and the Lhx2 factor are key in creating new hair follicles.

Scarring alopecia affects different hair follicle stem cells than nonscarring alopecia, and the infundibular region could be a new treatment target.