Search

everythinglearnresearchcommunity

for

Search:↓

Activating the Nrf2 pathway reduces inflammation and cell activation in human hair follicles, suggesting a potential treatment for certain hair loss conditions.

Eating glucoraphanin can help prevent psychosis in offspring whose mothers had immune system activation.

Increased PPARGC1α relates to hair thinning in common baldness.

RANK is a key target in breast cancer treatment due to its role in tumor growth and bone metastasis.

Most hair follicle stem cells do not protect their DNA by dividing it unevenly.

Understanding the immune-related causes of Alopecia Areata has led to potential treatments like JAK inhibitors.

Blue-light activation of TrkA improves hair-follicle stem cells' ability to become neurons and glial cells.

Muscle around hair follicles controls hair loss by releasing a signal that causes cell death.

Blocking cell death in hair follicles can lead to impaired hair growth.

Activating β-catenin in certain skin cells speeds up hair growth in mice.

Activating Wnt in skin cells controls the number of hair follicles by directing cell movement and fate.

Aging reduces stem cell activation, leading to hair loss in mice lacking a specific enzyme.

Activating STAT5 in the skin's dermal papilla is key for starting hair growth, regenerating hair follicles, and healing wounds.

Blocking β-catenin in skin cells improves hair growth during wound healing.

The study found that immune responses disrupt hair growth cycles, causing hair loss in alopecia areata.

Integrin alphavbeta6 is important for wound healing and hair growth, and blocking it may improve these processes.

Activating TERT in mice skin boosts hair growth by waking up hair follicle stem cells.

Old people have less hair because their hair follicles don't regenerate as well, not because of fewer stem cells, and a protein called follistatin might help reactivate hair growth.

Activating β-catenin in different skin stem cells causes various types of hair growth and skin tumors.

A synthetic sandalwood odorant can boost antimicrobial production in hair follicles, making them more resistant to bacteria.

The NF-κB effector p65/RelA activates hair keratin genes, aiding hair formation.

Activating Nrf2 helps wounds heal faster by increasing hair follicle stem cells.

Activating TLR3 helps improve skin and hair follicle regeneration after wounds.

TLR3 activation helps improve skin and hair follicle healing in mice.

Hair follicle cells help protect against immune attacks by regulating T-cell activity.

PTEN helps control the number and health of skin stem cells by working with the protein BMAL1.

Wnt/ß-catenin signaling is crucial for regulating skin stem cells and hair growth, with the right levels and timing needed for proper function.

c-Myc activation in mouse skin increases sebaceous gland growth and affects hair follicle development.

Nuclear Factor I-C is important for controlling hair growth by affecting the TGF-β1 pathway.

GLI2 increases follistatin production in human skin cells.