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Research 13
- Protein Kinase C Downregulation upon Rapamycin Treatment Attenuates Neuroinflammation and Mitochondrial Disease
- ROR2 regulates self-renewal and maintenance of hair follicle stem cells
- Protein kinase C is a key target for attenuation of Leigh syndrome by rapamycin
- 12-O-Tetradecanoylphorbol-13-acetate Induces Apoptosis in Renal Epithelial Cells through a Growth Signal Conflict Which Is Prevented by Activated ras1
- Retinoid-mediated stimulation of steroid sulfatase activity in myeloid leukemic cell lines requires RARα and RXR and involves the phosphoinositide 3-kinase and ERK-MAP kinase pathways
- Bisindolylmaleimide Protein-Kinase-C Inhibitors Delay the Decline in DNA Synthesis in Mouse Hair Follicle Organ Cultures
- Protein Kinase C as a Therapeutic Target
- Two distinct signaling pathways in hair cycle induction: Stat3-dependent and -independent pathways
- Two Mechanisms Regulate Keratin K15 Expression In Keratinocytes: Role of PKC/AP-1 and FOXM1 Mediated Signalling
- PKC downregulation upon rapamycin treatment attenuates mitochondrial disease
- Wnt/β-Catenin Signaling Stabilizes Hemidesmosomes in Keratinocytes
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