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Dying cells can help with faster healing and new hair growth by releasing a growth-promoting molecule.

Lactate production is important for activating hair growth stem cells.

Inactive hair follicle stem cells help prevent skin cancer.

Deleting the BRD4 protein in certain skin cells causes hair loss and skin inflammation.

Aging reduces skin cell renewal and defense against germs due to TGFbeta, but blocking TGFbeta could help restore these functions.

MicroRNA-205 helps hair grow by changing the stiffness and contraction of hair follicle cells.

The document concludes that the development of certain tumors is influenced by genetic background and that a specific gene modification can lead to tumor regression and reduced growth.

β-catenin in the dermal papilla is crucial for normal hair growth and repair.

The skin and thymus develop similarly to protect and support immunity.

STAT3 signaling is important for healthy skin and hair follicles, and its disruption can lead to skin conditions like atopic dermatitis.

Thymic stromal lymphopoietin (TSLP) promotes hair growth, especially after skin injury.

Dermal fibroblasts have adjustable roles in wound healing, with specific cells promoting regeneration or scar formation.

Removing Mediator 1 from certain mouse cells causes teeth to grow hair instead of enamel.

Skin bacteria, specifically Staphylococcus aureus, help in wound healing and hair growth by using IL-1β signaling. Using antibiotics on skin wounds can slow down this natural healing process.

Proteins like aPKC and PDGF-AA, substances like adenosine and ATP, and adipose-derived stem cells all play important roles in hair growth and health, and could potentially be used to treat hair loss and skin conditions.

Glutamine metabolism affects hair stem cell maintenance and their ability to change back to stem cells.

The TCL1 transgenic mouse model is useful for understanding human B-cell leukemia and testing new treatments.

TPA promotes hair growth by increasing stem cell activity and activating specific cell signals.

The research found that different types of fibroblasts are involved in wound healing and that some blood cells can turn into fat cells during this process.

Stem cells help heal skin wounds by moving and changing roles, working with other cells, and needing more research on their activation and behavior.

A WNT10A gene mutation leads to ectodermal dysplasia by disrupting cell growth and differentiation.

Mice without the Sept4/ARTS gene heal wounds better due to more stem cells that don't die easily.

Research on epidermal stem cells has advanced significantly, showing promise for improved clinical therapies.

Hormones during pregnancy and lactation keep skin stem cells inactive, preventing hair growth.

The protein SLC1A3 is important for activating skin stem cells and is necessary for normal hair and skin growth in mice.

The article concludes that hair loss is a common side effect of drugs treating skin cancer by blocking the hedgehog pathway, but treatment should continue, and more selective drugs might prevent this side effect.

In vivo lineage labelling is better than in vitro methods for identifying and understanding stem cells.

New treatments targeting the Hedgehog pathway can help treat advanced skin cancer but may have side effects and their effectiveness in early stages is unknown.

Stem cells compete for space using cell adhesion, and mutations can affect their competitive success, with implications for tissue health and disease.

Celsr1 is crucial for skin cell alignment, while Celsr2 has little effect on this process.