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Overactivating Hedgehog signaling makes hair follicle cells in mice grow hair faster and create more follicles.

The document concludes that careful design of genetic fate mapping experiments is crucial for accurate cell lineage tracing in mice.

Stem cell niches are crucial for regulating stem cell behavior and tissue health, and their decline can impact aging and cancer.

The method allows for 3D tracking of hair follicle stem cells and shows they can regenerate hair for up to 180 days.

Skin lesions in Carney complex are likely caused by a specific group of skin cells that promote pigment production due to a genetic mutation.

Basal cell carcinoma mostly starts from cells in the upper skin layers, not hair follicle stem cells.

Different small areas within hair follicles send specific signals that control what type of cells stem cells become.

Skin lesions in Carney Complex are caused by a gene change in some skin cells that leads to increased pigmentation and may lead to tumors.

Dermal Wnt/β-catenin signaling is important for the proper size and development of hair follicles.

New research suggests that skin cell renewal may not require a special type of cell previously thought to be essential.

Stem cell self-renewal is complex and needs more research for full understanding.

Compartmentalized organization might be crucial for stem cells to effectively respond to growth or injury.

Different types of stem cells with unique roles exist in blood, skin, and intestines, and this variety is important for tissue repair.

A specific group of slow-growing stem cells marked by Thy1 is crucial for skin maintenance and healing in mice.

CD4+ skin cells may be precursors to basal cell carcinoma.

Increasing Rps14 helps grow more inner ear cells and repair hearing cells in baby mice.

Epidermal stem cells are diverse and vary in activity, playing key roles in skin maintenance and repair.

Melanocyte development from neural crest cells is complex and influenced by many factors, and better understanding could help treat skin disorders.

Adipocytes can change into fibroblast-like cells to help with wound healing.

Skin-derived precursors in hair follicles come from different origins but function similarly.

Fibroblast state switching is crucial for skin healing and development.

Skin cancer often starts from Lgr5+ progenitor cells.

Genetic mutations can cause hair growth disorders by affecting key genes and signaling pathways.

Skin cell types develop when specific genes are turned on by removing certain chemical tags from DNA.

Adult esophageal cells can start to become like skin cells, with a key pathway influencing this change.

Genetic mutation and carcinogen treatment are both needed for skin cancer to develop in these specific mice.

The Megsin-Cre transgene is a new tool for genetic manipulation in the skin and upper digestive tract.

Different types of skin cells have unique genetic markers that affect how likely they are to spread cancer.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

Stem cells control their future role by changing ERK signal timing, affecting tissue regeneration and cancer.