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AIRE protein, defective in APECED patients, is found in skin and hair cells and interacts with cytokeratin 17.

The protein p53 directly reduces the production of Keratin 17, a skin and hair protein, in rats with radiation dermatitis.

Doctors should actively prevent and treat skin side effects in cancer patients to keep them on the best medication and reduce discomfort.

Certain genetic changes in the LSS gene cause a rare skin and hair condition.

A married couple both developed a rare type of hair loss, possibly due to shared environmental factors.

RSPO1 mutations in certain patients lead to skin cells that don't develop properly and are more likely to become invasive, increasing the risk of skin cancer.

Yellow dots and short vellus hairs are the most common signs of Alopecia Areata (AA), and trichoscopy can help diagnose AA and track treatment progress.

A specific mutation in the K25 gene causes a rare genetic disorder with curly hair at birth and later hair loss, along with dental issues.

A rare hair follicle disorder can cause itchy, bluish-black bumps on the body.

A specific hair protein variant increases the spread of breast cancer and is linked to worse survival rates.

c-Myc activation in mouse skin increases sebaceous gland growth and affects hair follicle development.

The absence of functional sebaceous glands causes hair follicle destruction and scarring alopecia.

Mutations in the Vitamin D receptor gene can cause hair loss similar to mutations in the Hairless gene.

Human hair keratin can be used in many medical applications.

Scientists successfully created mouse hair proteins in the lab, which are stable and similar to natural hair.

Early intervention and tailored management can reduce skin side effects from cancer treatments.

Trichohyalin-like proteins are essential for the development of skin structures like hair, nails, and feathers.

Oral isotretinoin may slightly improve acne but increases the risk of side effects like dry lips and skin; more research is needed to understand its full risks and benefits.

Lack of Ctip2 in skin cells delays wound healing and disrupts hair follicle stem cell markers in mice.

Understanding hair follicles through various models can help develop new treatments for hair disorders.

Fingerprints form uniquely before birth due to specific genetic pathways and local signals.

Clim proteins are essential for maintaining healthy corneas and hair follicles.

Frizzled 3 and Frizzled 6 together control the orientation of mouse hair follicles.

Keratin from hair binds well to gold and BMP-2, useful for bone repair.

A gene variant increases the risk of a type of hair loss by affecting hair protein production.

Platelet-rich plasma shows potential for hair loss and skin rejuvenation but needs more research for widespread use.

New treatments like nanotechnology show promise in improving skin cancer therapy.

Basonuclin 2 is vital for the development of facial bones, hair follicles, and male germ cells in adult mice, and its absence can lead to dwarfism and abnormal follicles.

Bmpr2 and Acvr2a receptors are crucial for hair retention and color.

K42 and K124 keratins are only found in horse hoof lamellae.