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Genetically modified stem cells from human hair follicles can lower blood sugar and increase survival in diabetic mice.

Cholesterol-related compounds can stop hair growth and cause inflammation in a type of scarring hair loss.

HIV-1 may cause increased stem cell death in hair follicles, leading to hair loss.

A gene called Gk5 controls lipid production in the skin and affects hair growth.

Cholecystokinin may help reduce skin inflammation in psoriasis.

HPH-15, a new compound, effectively reduces skin fibrosis in experiments without causing harm.

Topical glucocorticoids thin the skin and change collagen structure.

Formulation P-08-016 better targets hair follicles for baldness treatment.

Neuregulin3 affects cell development in the skin and mammary glands.

Certain mouse strains develop a skin condition similar to a human hair loss disease due to genetic defects.

Sub3 is essential for fungus adherence but not for skin invasion.

Human skin cells with stem-like features can help create new hair follicles and sebaceous glands when combined with other cells.

Mutations in NIPAL4 cause skin issues by disrupting lipid layers, but some improvement is seen with topical treatment.

Human dermal stem cells can become functional skin pigment cells.

Palmitoyl tetrapeptide-20 may help reduce hair greying and increase melanin production.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

The enzyme pyruvate kinase M2 helps hair regrowth and could be a potential treatment for hair loss.

Mice genetically modified to produce more CD109 in their skin had less inflammation and better healing with less scarring.

Paeonia lactiflora and Poria cocos extracts can potentially increase hair growth and reduce hair loss symptoms by lowering testosterone and inflammation levels.

The mTOR pathway proteins are altered in the hair follicles of patients with Lichen Planopilaris and Frontal Fibrosing Alopecia.

Innate lymphoid cells help control skin bacteria by regulating sebaceous glands.

Wound healing can help prevent hair loss from chemotherapy in young rats by increasing interleukin-1β signaling.

Transplanted hair follicles can change and adapt to new areas of the body, with the immune system possibly playing a role in this adjustment.

Integrin β1 is crucial for liver structure and function, preventing fibrosis.

The method increases stem-like cells for better skin regeneration.

Minor injuries to hair follicles can stimulate hair growth in mice by increasing a specific protein.

Killing specific cells in hair follicles can lead to hair growth problems in mice.

Smad4 is important for healthy hair follicles because it helps produce a protein needed for hair to stick together and grow.

Deep phenotyping helps distinguish between xeroderma pigmentosum and trichothiodystrophy, aiding in diagnosis and treatment.

Too much Smad7 changes skin and hair development by breaking down a protein called β-catenin, leading to more oil glands and fewer hair follicles.