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The conclusion suggests that PCOS may persist due to genetic traits that, while harmful for female fertility, could have provided survival and reproductive benefits to males.

Effective treatments for spinal and bulbar muscular atrophy are not yet available; more research is needed.

Multimodal primary treatment improves survival in premenopausal breast cancer patients and is also beneficial for postmenopausal women.

Excess androgens may cause PCOS, not just be a symptom.

The conclusion is that endocrinology significantly impacts medicine with various common medications used for treatment.

Hair growth is influenced by hormones and goes through different phases; androgens can both promote and inhibit hair growth depending on the body area.

Amenorrhea is relatively rare and initial testing should check FSH, TSH, and prolactin levels.

Hormones are crucial for regulating body functions and imbalances can lead to health issues.

Finasteride can have lasting negative effects on brain function and behavior by disrupting neurosteroid production.

Melasma is a skin condition linked to female hormones, genetics, UV exposure, and certain medications, but not to pituitary, adrenal, or thyroid diseases.

Hormonal therapy is a treatment option for acne, the only medical treatment for hirsutism, and the most promising for androgenetic alopecia.

Menopause causes significant changes in the vaginal introitus, but less so in the labia majora, which may lead to symptoms of vaginal atrophy.

Anabolic steroids boost muscle growth, SARMs increase muscle mass and bone density without side effects, and myostatin inhibitors block a protein that stops muscle growth, but each has potential risks.

Hormone therapy improves mental well-being in transgender individuals but requires ongoing health monitoring.

Choosing the right starting dose of Clomiphene Citrate for ovulation induction in PCOS women is challenging; higher BMI and hormone levels may indicate resistance to the standard dose.

Certain brain hormones and chemicals are linked to the development of Polycystic Ovary Syndrome.

New treatments are needed for PCOS that target its genetic, hormonal, and metabolic causes.

Genes and epigenetic changes are important in the development of Polycystic Ovary Syndrome.

Understanding genes and hormones is crucial for managing male puberty and sex development disorders.

TNFα, IFNγ, and Substance P significantly affect prolactin levels in human skin, suggesting new treatments for skin and hair conditions.

Some lesser-known causes of PCOS include autoimmune issues, genetic mutations, and changes in the body's microbiome.

Non-hormonal treatments should be used first for sexual dysfunction in postmenopausal breast cancer patients on aromatase inhibitors, with hormones as a second option.

PCOS causes infertility mainly due to hormonal imbalances, insulin resistance, and chronic inflammation.

Microglia, the brain's immune cells, may contribute to Polycystic Ovary Syndrome (PCOS) by altering the female brain's structure and function, with kisspeptin neurons and GABA neurotransmitters also playing a role.

Lifestyle changes are the first step in treating infertility in PCOS, followed by medications like clomiphene and metformin, and then surgery if needed.

Molecular diagnostics help identify genetic defects causing endocrine diseases, improving diagnosis and treatment options.

Selenium might help with insulin resistance and cholesterol in PCOS, but more research is needed to confirm its benefits.

Finasteride negatively affects fish reproduction and gonadal development.

Human alpha defensin 5 helps heal wounds, reduce bacteria, and grow hair on burned skin.

A boy with a rare form of early puberty caused by a new gene mutation responded well to treatment aimed at reducing testosterone and preserving adult height.