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- 865 Frontal fibrosing alopecia is associated with dysregulation of cholesterol biosynthesis pathways, fibrosis and mast cells
- 923 Molecular phenotyping of lichen planopilaris revealed dysregulation of cholesterol/fatty acid metabolism, fibrosis and mast cell pathways
- Interleukin‐10 Derived Apoptotic Vesicles Enhance Scarless Skin Healing by Modulating Fibroblast Metabolism and Fibrosis Pathways
- The Significance of NOTCH Pathway in the Development of Fibrosis in Systemic Sclerosis
- Exploring the Therapeutic Potential of TGF-β Inhibitors for Liver Fibrosis: Targeting Multiple Signaling Pathways
- 1493 Dynamic changes in fibroblast subpopulations drives development of radiation-induced skin fibrosis through the fra/c-jun pathway
- Wedelolactone Attenuates Pulmonary Fibrosis Partly Through Activating AMPK and Regulating Raf-MAPKs Signaling Pathway
- SIRT1-modified human umbilical cord mesenchymal stem cells ameliorate experimental peritoneal fibrosis by inhibiting the TGF-β/Smad3 pathway
- 863 Central centrifugal cicatricial alopecia gene expression analysis revealed cholesterol, fatty acid, and mast cell pathways
- LB1027 Identifying the mechanism of tension-associated tissue fibrosis based on single cell RNA sequencing on keloid scar
- The HEDGEHOG-GLI1 pathway is important for fibroproliferative properties in keloids and as a candidate therapeutic target
- Inhibition of Wnt/β‐catenin pathway promotes regenerative repair of cutaneous and cartilage injury
- WISP-1 induced by mechanical stress contributes to fibrosis and hypertrophy of the ligamentum flavum through Hedgehog-Gli1 signaling
- Tissue engineering strategies combining molecular targets against inflammation and fibrosis, and umbilical cord blood stem cells to improve hampered muscle and skin regeneration following cleft repair
- Primary cicatricial alopecias are characterized by dysregulation of shared gene expression pathways
- Dermal white adipose tissue: Development and impact on hair follicles, skin defense, and fibrosis
- Perifollicular Inflammation and Fibrosis in Androgenetic Alopecia: Implications for Diagnosis and Treatment - A Comparative Histopathologic and Clinical Study with Normal-Appearing Scalp
- Perifollicular Inflammation and Fibrosis in Androgenetic Alopecia: Implications for Diagnosis and Treatment - A Comparative Histopathologic and Clinical Study with Normal-Appearing Scalp
- The Role of Fibrosis in Androgenetic Alopecia: Mechanisms and Implications
- Supplementary Material for: The Role of Fibrosis in Androgenetic Alopecia: Mechanisms and Implications
- Supplementary Material for: The Role of Fibrosis in Androgenetic Alopecia: Mechanisms and Implications
- 3: Multimodal Molecular Analysis Reveals Divergent Trajectories Of Wound Regeneration Versus Fibrosis
- 711 Small molecule wnt pathway inhibitor (SM04755) as a potential topical psoriasis treatment
- Keloid pathogenesis and fibroproliferative properties are dependent on stem cells regulated by the HEDGEHOG-GLI1 pathway
- Proteomic Profiling of Central Centrifugal Cicatricial Alopecia Reveals Role of Humoral Immune Response Pathway and Metabolic Dysregulation
- IL-17 Expression in the Perifollicular Fibrosis in Biopsies From Lichen Planopilaris
- Wnt/β‐catenin pathway in tissue injury: roles in pathology and therapeutic opportunities for regeneration
- [Research advances on the mechanism of Wnt/β-catenin signaling pathway in body surface wound healing].
- [Influence and mechanism of extracellular vesicles derived from human dermal papilla cells on skin fibrosis in mice].
- Fibroblast heterogeneity: implications for human disease