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Fetal skin heals without scarring due to unique cells and processes not present in adult skin healing.

Certain signals and genes play a key role in hair growth and regeneration, and understanding these could lead to new treatments for skin regeneration.

The shape of fibrous scaffolds can improve how stem cells help heal skin.

Hair follicle stem cells are promising for wound healing but require more research for safe clinical use.

Mast cells and VEGF contribute to post-surgery adhesions, and blocking VEGF can reduce these adhesions; also, certain factors affect wound healing and fetal skin heals differently with age.

Blocking the Wnt/β‐catenin pathway can speed up wound healing, reduce scarring, and improve cartilage repair.

Careful selection of mice by genetics and age, and controlled housing conditions improve the reliability of hair regrowth in wound healing tests.

Different immune cells like platelets, mast cells, neutrophils, macrophages, T cells, B cells, and innate lymphoid cells all play roles in skin wound healing, but more research is needed due to inconsistent results and the complex nature of the immune response.

Fat-derived stem cells show promise for treating skin issues and improving wound healing, but more research is needed to confirm the best way to use them.

Most vertebrates can regenerate skin, nails, and corneas, but only some can regenerate teeth and lenses.

Overexpressing follistatin in mice delays wound healing and reduces scar size.

The document concludes that more research is needed on making and understanding biomaterial scaffolds for wound healing.

A certain type of skin cell, marked by EGFR, produces a lot of IGF1 and helps hair follicles grow back faster.

The hydrogel made of chitosan, HPMC, and insulin speeds up wound healing and could be a new dressing, especially for diabetics.

4-Aminopyridine improves skin wound healing and tissue regeneration by increasing cell growth and promoting nerve repair.

Stem cells are crucial for wound healing and understanding their role could lead to new treatments, but more research is needed to answer unresolved questions.

Mast cells likely promote skin scarring and fibrosis, but their exact role is still unclear.

Leptin-deficient mice, used as a model for Type 2 Diabetes, have delayed wound healing due to impaired contraction and other dysfunctional cellular responses.

A topical BRAF inhibitor, vemurafenib, can speed up wound healing and promote hair growth, especially in diabetic patients.

Artificial dermal template treatment can stimulate complete skin and hair follicle regrowth.

Different scaffold patterns improve wound healing and immune response in mouse skin, with aligned patterns being particularly effective.

γδ T cells help with hair growth during wound healing in mice.

Li2CO3 improved skin disease in a mouse model of Focal Dermal Hypoplasia without toxicity.

Wnt and Notch signaling help wound healing by promoting cell growth and regulating cell differentiation.

Spiny mice are better at regenerating hair after injury than laboratory mice and could help us understand how to improve human skin repair.

PI3K/Akt pathway is crucial for hair growth and regeneration.

Trichostatin A helps restore hair-growing ability in skin cells used for hair regeneration.

Mice genetically modified to produce more Del1 protein had faster hair regrowth.

Hydroxypinacolone retinoate is a potent anti-aging ingredient for skin that is more effective and less irritating than other forms of retinoids.

Fzd2 is important for skin and hair development through various signaling ways.