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Research 61–90 of 1000+
- Advancing Design Strategy of PROTACs for Cancer Therapy
- Gender-linked differences in human skin
- Steroid Hormones of Reproduction and Sexual Development
- Androgen Treatment in Women
- PCOS and Obesity: Contraception Challenges
- Evaluation and Treatment of Gender Dysphoria to Prepare for Gender Confirmation Surgery
- Hormonal contraceptives: pharmacology tailored to women's health
- Steroid Hormones
- TGFβ and cancer metastasis: an inflammation link
- Amphiregulin in Fibrotic Diseases and Cancer
- Skin Ageing in Women: Pathophysiology and Treatment
- Evaluation and Treatment of Hirsutism in Premenopausal Women: An Endocrine Society Clinical Practice Guideline
- Melatonin in the skin: synthesis, metabolism and functions
- Signal integrator function of CXXC5 in Cancer
- Gonads
- Glibenclamide inhibits cell growth by inducing G0/G1 arrest in the human breast cancer cell line MDA-MB-231
- The postnatal offspring of finasteride-treated male rats show altered ERα and PCNA expression in the liver
- <i>In-Vitro</i> Effect of Sex Steroids on Mouse Melanoma (B16F10) Cell Growth
- Anabolic androgenic steroids and carcinogenicity focusing on Leydig cell: a literature review
- NDA Submission of Vepdegestrant (ARV-471) to U.S. FDA: The Beginning of a New Era of PROTAC Degraders
- Dihydrotestosterone induces arterial stiffening in female mice
- Nitric Oxide: Physiological Functions, Delivery, and Biomedical Applications
- Bioavailability File: Bicalutamide
- Clinical use of 5α-reductase inhibitors
- Chemotherapy of psoriasis and other skin disorders with oral retinoids
- Current Management and Treatment Options for Breast Pain
- 2-Substituted 4-(Thio)chromenone 6-<i>O</i>-Sulfamates: Potent Inhibitors of Human Steroid Sulfatase
- Success and Controversy of Natural Products as Therapeutic Modulators of Wnt Signaling and Its Interplay with Oxidative Stress: Comprehensive Review Across Compound Classes and Experimental Systems
- Sex hormones and related compounds, including hormonal contraceptives
- Aromatase-independent testosterone conversion into estrogenic steroids is inhibited by a 5α-reductase inhibitor