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Minoxidil, a common alopecia medication, might cause eye changes due to its properties and lack of tissue selectivity.

A device was made in 2008 to measure hair loss severity. Other findings include: frizzy mutation in mice isn't related to Fgfr2, C/EBPx marks preadipocytes, Cyclosporin A speeds up hair growth in mice, blocking plasmin and metalloproteinases hinders healing, hyperbaric oxygen helps ischemic wound healing, amniotic membranes heal wounds better than polyurethane foam, rhVEGF165 from a fibrin matrix improves tissue flap viability and induces VEGF-R2 expression, and bFGF enhances wound healing and reduces scarring in rabbits.

Ovol2 is important for proper skin healing and hair growth.

Triparanol therapy can cause hair loss and skin dryness without inflammation or damage to hair follicles or skin structures.

Blocking a specific protein signal can make hair grow on mouse nipples.

A new treatment using AGED to modulate PPAR-γ shows promise for treating scarring hair loss by protecting and repairing hair follicle cells.

Hair regeneration needs dynamic cell behavior and mesenchyme presence for stem cell activation.

KGF and activin are crucial for skin healing and repair.

Oral retinoids are effective for various skin conditions but have side effects and should not be used during pregnancy.

Disruptions in hair follicle fibroblast dynamics can cause hair growth problems.

Scarring alopecias are complex hair loss disorders that require early treatment to prevent permanent hair loss.

Hair follicles can help wounds heal faster and this knowledge could be used to treat chronic skin ulcers, with a potential use of a special stem cell hydrogel to enhance healing.

Adding human blood vessel cells to hair follicle germs may improve hair growth and quality.

The protein ARA70/ELE1 is involved in male pattern baldness, and lower levels of its short form may lead to hair thinning.

Jagged-1 in skin Tregs is crucial for timely wound healing by recruiting specific immune cells.

Activating Wnt in skin cells controls the number of hair follicles by directing cell movement and fate.

Cicatricial alopecia, a permanent hair loss condition, is mainly caused by damage to specific hair follicle stem cells and abnormal immune responses, with gene regulator PPAR-y and lipid metabolism disorders playing significant roles.

After skin injury, adult mice can grow new hair follicles, and this process can be increased or stopped by manipulating Wnt signals.

Understanding where cancer cells come from helps create better prevention and treatment methods.

Stem cells in the hair follicle are regulated by their surrounding environment, which is important for hair growth.

Estrogen Receptor ß (ERß) is the main hormone controller in human skin and hair follicles, not Estrogen Receptor α (ERα) or the Androgen Receptor (AR).

Cytokeratin 19 and cytokeratin 15 are key markers for monitoring the quality and self-renewing potential of engineered skin.

Human beard hair medulla contains a unique and complex mix of keratins not found in other human tissues.

EdnrB signaling helps melanocyte stem cells regenerate and could be targeted to treat pigmentation issues.

Tooth papilla cells can help regenerate hair follicles and grow hair.

Stem cells, especially from fat tissue and Wharton's jelly, can potentially regenerate hair follicles and treat hair loss, but more research is needed to perfect the treatment.

The enzyme that changes testosterone to a stronger form is mostly found in the part of the hair follicle called the dermal papilla.

Using fat stem cells and blood cell-rich plasma together improves healing in diabetic wounds by affecting cell signaling.

Skin and hair can regenerate after injury due to changes in gene activity, with potential links to how cancer spreads. Future research should focus on how new hair follicles form and the processes that trigger their creation.

IGF-1 increases whisker growth in transgenic mice.