Search

everythinglearnresearchcommunity

for

Search:↓

Boosting certain cell signals can prevent hair loss from chemotherapy and radiation.

Boosting certain cell signals can prevent hair loss from chemotherapy and radiation.

M-CSF-stimulated myeloid cells can turn into skin cells and help heal wounds and regrow hair.

PRC2 is essential for maintaining intestinal cell balance and aiding regeneration after damage.

A specific group of skin stem cells was found to help maintain hair follicle cells.

Keeping β-catenin levels high in mammary cells disrupts their development and branching.

Odontogenic keratocysts are caused by abnormal Hedgehog signaling and can lead to tooth and bone issues.

TGM3 is important for skin and hair structure and may help diagnose cancer.

BLIMP1 is essential for skin maintenance but not for defining sebaceous gland progenitors.

The mTurq2-Col4a1 mouse model shows that cells can divide while attached to stable basement membranes during development.

Adding hyaluronic acid helps create larger artificial hair follicles in the lab.

The research found a new way to heal chronic skin ulcers by turning certain cells into skin tissue using specific factors.

Hair follicle development is controlled by interactions between skin tissues and specific molecular signals.

Hair growth phase and certain genes can speed up wound healing, while an inflammatory mediator can slow down new hair growth after a wound. Understanding these factors can improve tissue regeneration during wound healing.

Wnt1/βcatenin signaling is crucial for heart repair after injury.

Stem cells in the hair follicle are regulated by their surrounding environment, which is important for hair growth.

TGF-β signaling is crucial for stem cell maintenance, differentiation, and has implications for cancer treatment.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

Research on epidermal stem cells has advanced significantly, showing promise for improved clinical therapies.

Using fat stem cells and blood cell-rich plasma together improves healing in diabetic wounds by affecting cell signaling.

Human hair follicle dermal cells can effectively replace other cells in engineered skin.

Transit-amplifying cells are crucial for tissue repair and can contribute to cancer when they malfunction.

Hairless protein is crucial for healthy skin and hair, and its malfunction can cause hair loss.

The document concludes that while lab results for hair growth promotion are promising, human trials are needed and better testing methods should be developed.

The protein Par3 is crucial for healthy skin, affecting the skin barrier, cell differentiation, and stem cell maintenance.

HPV16 oncogenes disrupt the normal activity of hair follicle stem cells.

The gene Msx2 is crucial for hair follicle regeneration during wound healing.

Older people's sweat glands are less effective at helping skin wounds heal due to weaker cell connections.

Stem cells can help regenerate hair follicles.

Hair in mammals likely evolved from glandular structures, not scales.