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Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Neuregulin3 affects cell development in the skin and mammary glands.

The document concludes that hair curvature can be explained by the growth patterns caused by the shape and separation of cells in the hair follicle and is affected by specific molecular pathways.

Scientists have found a way to create hair follicles from human stem cells, which could potentially be used to treat hair loss.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

The document concludes that the skin's immune system is complex, involving interactions with hair follicles, nerves, and microbes, and can protect or cause disease, offering targets for new treatments.

Improving the environment and cell interactions is key for creating human hair in the lab.

Researchers isolated a new type of stem cell from mouse skin that can renew itself and turn into multiple cell types.

Nestin marks cells that can become a specific type of skin cell in hair follicles of both developing and adult mice.

Understanding gene expression in hair follicles can reveal insights into hair growth and disorders.

Wnt signaling is crucial for pigmented hair regeneration by controlling stem cell activation and differentiation.

Different types of skin cells have unique genetic markers that affect how likely they are to spread cancer.

The dermal papilla is crucial for hair growth and health, and understanding it could lead to new hair loss treatments.

The document concludes that while significant progress has been made in understanding skin biology and stem cells, more research is needed to fully understand their interactions with their environment.

The TGF-β family helps control how cells change and move, affecting skin, hair, and organ development.

The Ovol2-Zeb1 circuit is crucial for skin healing and hair growth by guiding cell movement and growth.

Too much β-catenin activity can mess up the development of mammary glands and make them more like hair follicles.

Keratin-associated proteins have ancient origins and were used for different purposes before being adapted for hair in mammals.

Nestin-expressing cells turn into a specific type of skin cell in hair follicles during development and in adults.

CDP is crucial for lung and hair follicle cell development.

Nestin identifies specific progenitor cells in hair follicles that can become outer root sheath cells.

Different small areas within hair follicles send specific signals that control what type of cells stem cells become.

DNMT1 helps turn hair follicle stem cells into fat cells by blocking a specific microRNA.

The enzymes Tet2 and Tet3 are important for skin cell development and hair growth.

Runx1 affects hair growth, cancer development, and autoimmune diseases in epithelial tissues.

Healthy mitochondria in skin cells are essential for proper hair growth and skin cell interaction in mice.

Stress can cause early aging in certain skin cells, leading to problems with hair growth.

Skin cells show flexibility in healing wounds and forming tumors, with potential for treating hair disorders and chronic ulcers.

Hair and skin healing involve complex cell interactions controlled by specific molecules and pathways, and hair follicle cells can help repair skin wounds.