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The nucleus is key in controlling skin growth and repair by coordinating signals, gene regulators, and epigenetic changes.

SOX9 helps determine stem cell roles by interacting with DNA and proteins that control gene activity.

Short-chain fatty acids from *Cutibacterium acnes* cause skin inflammation, contributing to acne.

Feather patterns form through genetic and epigenetic controls, with cells self-organizing into periodic patterns.

DNA methylation changes in women with PCOS could be used as disease markers and suggest new treatment targets.

Epigenetic factors play a crucial role in skin health and disease.

Tissue environment greatly affects the unique epigenetic makeup of regulatory T cells, which could impact autoimmune disease treatment.

Keratinocytes help heal skin wounds by interacting with immune cells and producing substances that kill pathogens.

The conclusion is that Treg-targeted therapies have potential, but more knowledge of Treg biology is needed for effective treatments, including for cancer.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

Valproic Acid could potentially be used to treat immune-related conditions due to its ability to modify immune cell functions.

Mice can grow new hair follicles after skin wounds through a process not involving existing hair stem cells, but requiring more research to understand fully.

Genetic research has advanced our understanding of skin diseases, but complex conditions require an integrative approach for deeper insight.

Feather patterns are influenced by enhancers and chromatin looping, and the structure of protein complexes important for hair growth has been detailed.

Environmental factors and exposure to toxins may contribute to male infertility by affecting sperm and hormone function.

Histone modification is key in treating chronic inflammatory skin diseases.

The symposium showed that stem cells are key for understanding and treating skin diseases and for developing new skin models and therapies.

Colorectal cancer development involves both genetic changes and epigenetic alterations like DNA methylation and microRNA changes.

Understanding phenotypic plasticity is crucial for developing effective cancer therapies.

Wnt-3a helps grow more skin stem cells, which could lead to new hair loss treatments.

The Pemphigus Disease Area Index and Autoimmune Bullous Skin Disorder Intensity Score are the best tools for assessing pemphigus severity.

Dermal fibroblasts have adjustable roles in wound healing, with specific cells promoting regeneration or scar formation.

Melanoma's complexity requires personalized treatments due to key genetic mutations and tumor-initiating cells.

New research shows that skin diversity is influenced by different types of dermal fibroblasts and their development, especially involving the Wnt/β-catenin pathway.

Human foreskin does not show aging or reduced cell growth after radiation, and H2A.J is not a good marker for radiation-induced aging.

Women with scarring alopecia are less likely to have used hormone replacement therapy or oral contraceptives compared to those with female pattern hair loss.

Histone demethylases play a key role in the development of many diseases and may be targets for treatment.

MOF controls key genes for skin development by regulating mitochondrial and ciliary functions.

Cancer can hijack the body's cell repair system to promote tumor growth, and targeting this process may improve cancer treatments.

Hoxc genes control hair growth through Wnt signaling.