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Rats can't grow new hair follicles after skin wounds, unlike mice, due to differences in gene expression and response to WNT signaling.

Acne inversa is an epithelial-driven disease where inflammation is caused by cyst rupture, and treatments should focus on preventing tendril growth for better results.

Different genes are active in dogs' hair growth and skin, similar to humans, which helps understand dog skin and hair diseases and can relate to human conditions.

UV exposure reduces Lgr6+ stem cells in mouse skin and they don't significantly contribute to skin cancer development.

Skin cells and certain hair follicle areas produce hemoglobin, which may help protect against oxidative stress like UV damage.

N-acetyl-L-cysteine (NAC) can prevent DNA damage and protect cells from harm.

Mouse tail skin has different keratinization near hair follicles and scales.

Overexpression of sPLA2-IIA in mouse skin reduces hair stem cells and increases cell differentiation through JNK/c-Jun pathway activation.

Hairless protein and putrescine regulate each other, affecting hair growth and skin balance.

Vitamin D receptor helps control hair growth and could be used to treat certain skin tumors.

Tiny infundibular cysts are the main lesions in chloracne, not comedones.

The first pediatric case of naevus trichilemmocysticus was documented.

A specific type of skin cell creates an opening for hair to grow out, and problems with this process can lead to skin conditions.

Overactive Wnt signaling in mouse skin stem cells causes acne-like cysts and shrinking oil glands, which some treatments can partially fix.

The cornified envelope is crucial for skin's barrier function and involves key proteins and genetic factors.

Decreased IGF-1R expression may contribute to sacrococcygeal pilonidal sinus development.

The Hairless gene is crucial for healthy skin and hair growth.

Skin changes throughout life, from development before birth to aging effects like wrinkles, influenced by both genetics and environment.

Stem cell offspring help control their parent stem cells, affecting tissue health, healing, and cancer.

Different types of skin cells play specific roles in development, healing, and cancer.

Oxidative stress contributes to hair graying and loss as we age.

Keratin proteins are crucial for healthy skin, but mutations can cause skin disorders with no effective treatments yet.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

Vitamin D receptor is crucial for starting hair growth after birth.

Wound dressing absorbs fluid, regenerates hair follicles, and heals skin burns.

BLIMP1 is essential for skin maintenance but not for defining sebaceous gland progenitors.

Disrupting Acvr1b in mice causes severe hair loss and thicker skin.

Eph/ephrin signaling is important for skin cell behavior and could be targeted to treat skin diseases.

The skin is the largest organ, protecting the body, regulating temperature, and producing hormones.

Keratin 17 is modified by RSK1 in response to growth and stress, affecting skin growth and stress response.