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Vitamin D may help protect against nonmelanoma skin cancer.

Overexpression of a specific receptor in mice skin causes skin thinning, early skin barrier formation, eye issues, and hair loss.

Corneous beta-proteins evolved uniquely in reptiles and birds, forming scales, claws, beaks, and feathers.

The oncoprotein causes abnormal hair growth without increasing skin cancer risk.

Pemetrexed is as effective as docetaxel but has fewer side effects for treating nonsmall-cell lung cancer after EGFR-TKI therapy failure.

Prednisone reduces organ mast cell infiltration but does not alter the abnormal appearance of mast cells in systemic mastocytosis.

Wnt4 protein makes the outer skin layer thicker in burn wounds by turning on a specific healing pathway and loosening the connections between skin cells.

Overexpressing ornithine decarboxylase and v-Ha-ras in keratinocytes leads to invasiveness and malignancy.

Cat color patterns are determined early in development by gene expression and epidermal changes, with the Dickkopf 4 gene playing a crucial role.

Epidermal stem cells maintain skin health through specific niches and signaling pathways.

Non-immune dermal cells dominate, epidermal cells increase after day 9, and certain immune cells persist beyond inflammation in wound-induced hair follicle regeneration.

Using DNase enzymes to break down harmful NETs could be a new treatment for Stevens-Johnson syndrome and toxic epidermal necrolysis.

Hair loss treatments show limited improvement, sunscreen thickness affects vitamin D production, and the effectiveness of IVIg for toxic epidermal necrolysis is uncertain.

Finasteride is effective for male hair loss, teledermatology is accurate and accepted, and cyclosporin helps treat toxic epidermal necrolysis.

Adult mouse skin contains stem cells that can create new hair, skin, and oil glands.

The document concludes that skin stem cells are important for hair growth and wound healing, and could be used in regenerative medicine.

The paper concludes that understanding melanocyte development can help in insights into skin diseases and melanoma diversity.

c-Myc activation in mouse skin increases sebaceous gland growth and affects hair follicle development.

High levels of ornithine decarboxylase can cause tumors in mouse skin.

Stem cells are crucial for skin repair and new treatments for chronic wounds.

Understanding where cancer cells come from helps create better prevention and treatment methods.

Activating the Sonic hedgehog gene in mice can start the hair growth phase.

Skin fat cells help with skin balance, hair growth, and healing wounds.

Stem cells help heal skin wounds by moving and changing roles, working with other cells, and needing more research on their activation and behavior.

Blocking testosterone speeds up wound healing in males.

Scientists have found a way to create hair follicles from skin cells of newborn mice, which can grow and cycle naturally when injected into adult mouse skin.

Visible light can damage skin and most sunscreens don't block it well; more research is needed on its effects and protection methods.

Targeted cancer therapies often cause serious skin problems that need careful management.

Epigenetic changes control how adult stem cells work and can lead to diseases like cancer if they go wrong.

Bone Morphogenetic Proteins (BMPs) help control skin health, hair growth, and color, and could potentially be used to treat skin and hair disorders.