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Nagashima-type palmoplantar keratosis in Asians is caused by a SERPINB7 gene mutation.

New findings suggest potential treatments for melanoma, hyperpigmentation, hair defects, and multiple sclerosis, and show skin microbiome changes don't cause atopic dermatitis.

Some cells may slow melanoma growth, a protein could affect skin pigmentation, a gene-silencing method might treat hair defects, skin bacteria changes likely result from eczema, and a defensin protein could help treat multiple sclerosis.

New treatments for skin conditions in children include a preferred drug for birthmark reduction, proactive creams for eczema and vitiligo, a safe psoriasis medication, and special tissues and socks for eczema and fungal infections.

Different types of cell death affect skin health and inflammation, and understanding them could improve treatments for skin diseases.

TGM3 is important for skin and hair structure and may help diagnose cancer.

Mice with extra human KLK14 had hair and skin problems, including weaker cell bonds and inflammation, linked to Netherton syndrome.

Orobanche rapum extract rejuvenates skin and protects skin bacteria, leading to healthier skin.

sPLA2-IIE is crucial for normal hair follicle structure and skin health.

Understanding keratinization is crucial for treating skin conditions like ichthyoses and psoriasis.

Cystatin M/E helps in the final stages of hair and nail formation by controlling certain enzymes.

Cornification is the process where living skin cells die to create a protective barrier, and problems with it can cause skin diseases.

Lack of cystatin M/E causes thin hair and dry skin.

Not having enough cystatin M/E protein causes less hair growth and dry skin.

Cholesterol sulfate buildup due to a genetic mutation disrupts the skin barrier, leading to the scaling skin seen in X-linked ichthyosis.

The extracellular matrix is crucial for controlling skin stem cell behavior and health.

Older skin has higher cancer risk due to inflammation and stem cell issues.

Epidermal keratinocytes start wound healing and inflammation after schistosome infection.

HSD11b1 affects skin nerves and increases non-histaminergic itch.

New treatments for skin and hair repair show promise, but further improvements are needed.

Eph/ephrin signaling is important for skin cell behavior and could be targeted to treat skin diseases.

NIR-II imaging effectively tracked stem cells that helped repair facial nerve defects in rats.

Mouse profilaggrin helps in skin cell differentiation and may be involved in calcium signaling.

Healthy skin prevents water loss and protects against threats.

VEGF gel increases hair growth but may have potential toxicity.

IL-1 family cytokines are crucial for skin defense and healing, but their imbalance can cause skin diseases.

The study created hair-bearing skin models that lack a key protein for skin layer attachment, limiting their use for certain skin disease research.

Human Schwann cells can be quickly made from hair follicle stem cells for nerve repair.

Cystatin M/E strongly inhibits cathepsin V and cathepsin L, important for skin formation.

Papain cream removes hair better than papain gel, causing hair follicles to widen and skin to thicken.