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Researchers found new hair and nail genes, how hair reacts to UV, differences in white and pigmented hair growth, nerve changes in alopecia, treatments for baldness and alopecia, a toenail condition linked to a genetic disorder, and that nail fungus is more common in people with psoriasis.

Cell death shapes skin stem cell environments, affecting inflammation, repair, and cancer.

The document concludes that understanding the genes and pathways involved in hair growth is crucial for developing treatments for hair diseases.

Foxi3 expression in developing teeth and hair is controlled by the ectodysplasin pathway.

High androgen levels and genetic factors likely cause Becker's nevus and related symptoms.

HPV genes and estradiol increase a cancer-related signaling pathway, which may be targeted for cervical cancer treatment.

Three dogs with a rare skin condition improved with treatment.

New treatments targeting specific genes show promise for treating keratin disorders.

Mice with LSS deficiency showed hair loss and cataracts, similar to humans, and can help in understanding and treating this condition.

The conclusion is that different types of hair loss in dogs and cats can be cosmetic or serious, and affected animals should not be bred.

First Korean case of Cronkhite-Canada syndrome with colon cancer and serrated adenoma.

The document concludes that while significant progress has been made in understanding skin biology and stem cells, more research is needed to fully understand their interactions with their environment.

Mice lacking Insig proteins had hair growth problems due to cholesterol buildup, but this was fixed by the drug simvastatin.

Patient-derived melanocytes can potentially treat vitiligo by restoring skin pigmentation.

The cornified envelope is crucial for skin's barrier function and involves key proteins and genetic factors.

Immune cells are essential for early hair and skin development and healing.

Human skin has multiple layers and functions, with key roles in protection, temperature control, and appearance.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Edar signaling is crucial for proper hair follicle development and function.

C/EBPalpha and C/EBPbeta are crucial for normal skin and oil gland cell development in adult mice.

A mouse gene mutation increases the risk of skin cancer.

Skin's epithelial stem cells are crucial for repair and maintenance, and understanding them could improve treatments for skin problems.

Blocking YAP/TAZ could be a new way to treat skin cancer.

Baricitinib and its combination with lonafarnib improve fat cell formation in certain genetic disorders.

Skin cancer often starts from Lgr5+ progenitor cells.

Keeping β-catenin levels high in mammary cells disrupts their development and branching.

Removing Mediator 1 from certain mouse cells causes teeth to grow hair instead of enamel.

Loss of TET2 increases the risk of skin and oral cancer.

Activating Nrf2 helps wounds heal faster by increasing hair follicle stem cells.

Understanding hair follicle development can help treat hair loss, skin regeneration, and certain skin cancers.