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The early genes of a specific virus can cause abnormal skin cell growth and hair follicle changes.

A virus protein can activate a pathway that may lead to abnormal hair follicle development.

Dermal Wnt/β-catenin signaling is important for the proper size and development of hair follicles.

The protein CTCF is essential for skin development, maintaining hair follicles, and preventing inflammation.

A gene deletion causes the "hairless" trait in Iffa Credo rats.

Cell death shapes skin stem cell environments, affecting inflammation, repair, and cancer.

Non-invasive methods can effectively diagnose and manage alopecia areata.

Cilostazol may help hair grow and could be a new treatment for hair loss.

The conclusion is that accurate diagnosis of different types of hair loss requires good teamwork between skin doctors and lab experts.

Hair growth-related cells need the enzyme SCD1 to help maintain the area that supports hair growth.

Certain microRNAs may protect against hair loss in alopecia areata and could be potential treatment targets.

Injecting platelet-rich plasma under the skin significantly improves hair growth and quality without harmful effects.

Four genes are potential markers for hair loss condition alopecia areata, linked to a specific type of cell death.

Scientists can control how skin stem cells divide by using different treatments.

Most people worried about abnormal hair loss actually have psychological issues like depression or anxiety.

Some breast cancer patients developed permanent hair loss after chemotherapy and hormonal therapy, showing patterns similar to common baldness and alopecia areata.

Trichotillomania is more common in young females and often linked to stress and psychiatric conditions, with hair loss and increased catagen hair observed.

Hair loss can significantly affect a person's self-esteem and body image, especially in young people, those who value their looks highly, and women.

MRI helps evaluate androgenetic alopecia severity.

Loose anagen hair syndrome is caused by structural abnormalities in the hair follicle's inner root sheath.

Acne lesions start with changes in hair follicles and increase in inflammation, suggesting a cycle that could affect treatment strategies.

Male pattern hair loss caused by follicular miniaturization; early diagnosis and treatment can reduce psychological burden.

Uncombable hair syndrome causes frizzy hair and can affect the nervous system, eyes, and ears, often co-occurring with other hair, skin, nail, and teeth conditions, and is linked to three specific gene mutations.

An 8-year-old girl has a rare genetic disorder causing complete, irreversible hair loss and skin bumps.

Spironolactone may help reduce hair loss in androgenic alopecia.

Treatments for hair loss include hormone modifiers, minoxidil, and hair transplant surgery.

Chronic Telogen Effluvium may resolve after years and is diagnosed by examining the patient's history and clinical signs, with treatment aimed at underlying causes and possibly minoxidil.

A 5-year-old boy's skin condition improved with systemic valganciclovir after a cardiac transplant and immunosuppressive therapy.

Identical twins had different symptoms because one had more cells with an extra chromosome fragment in different tissues.

Androgenetic alopecia is a common hair loss condition influenced by various factors and linked to psychosocial and cardiovascular issues.