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Research 241–270 of 1000+
- The Skin and Inflamm-Aging
- Aging skin and non-surgical procedures: a basic science overview
- Is There a Histone Code for Cellular Quiescence?
- Innate immunity and the regulation and mobilization of keratinocyte stem cells: are the old players playing a new game?
- The Potential Reversible Transition between Stem Cells and Transient-Amplifying Cells: The Limbal Epithelial Stem Cell Perspective
- Highlights from the Literature
- Calreticulin: non‐endoplasmic reticulum functions in physiology and disease
- Quiescent Tissue Stem Cells Evade Immune Surveillance
- Aging of the hair follicle pigmentation system
- Keratins as an Inflammation Trigger Point in Epidermolysis Bullosa Simplex
- Hypoxia in Cell Reprogramming and the Epigenetic Regulations
- Cell-Free Therapies: The Use of Cell Extracts to Mitigate Irradiation-Injured Salivary Glands
- Epidermal-Derived Hedgehog Signaling Drives Mesenchymal Proliferation during Digit Tip Regeneration
- Myrtus communis Leaf Extract Modulates IL-1α and VEGF Gene Expressions in HaCaT Cells: Probable Implications for Its Beneficial Effects in Hair Loss and Wound Healing
- Inhibition of the Hippo pathway by verteporfin reduces the proliferation and stemness of rat hair follicle neural crest stem cells under hypoxia
- 573 Hair follicles employ a 2-dimensional healing strategy to repair 3-dimensional injuries
- Interrelation of immunity and tissue repair or regeneration
- Flavonoids in safflower extract reduce cisplatin-induced damage to human follicle dermal papilla cells by inhibiting DNA damage and Rad17/Chk1/Cdc25C signaling
- Research progress on mechanisms of skin aging and anti-aging strategies
- Association of DNA damage with vitamin D and hair heavy metals of obese women
- Differential Expression of Fibromodulin, a Transforming Growth Factor-β Modulator, in Fetal Skin Development and Scarless Repair
- Androgen Receptor Accelerates Premature Senescence of Human Dermal Papilla Cells in Association with DNA Damage
- Hair Graying: Mechanisms, Prevention, and Innovative Treatment Strategies – literature review
- The role of nuclear hormone receptors in cutaneous wound repair
- Transient mitochondrial DNA double strand breaks in mice cause accelerated aging phenotypes in a ROS-dependent but p53/p21-independent manner
- Skin aging: mechanisms, evaluation, and rejuvenation
- EZH1 and EZH2 cogovern histone H3K27 trimethylation and are essential for hair follicle homeostasis and wound repair
- Development of a Chitosan Nanofibrillar Scaffold for Skin Repair and Regeneration
- Neural Mechanisms of Hair Growth Control
- Activin Controls Skin Morphogenesis and Wound Repair Predominantly via Stromal Cells and in a Concentration-Dependent Manner via Keratinocytes