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Research 151–180 of 158
- Semecarpus anacardium L. (Anacardiaceae)
- Conversion of C57Bl/6 mice from a tumor promotion-resistant to a -sensitive phenotype by enhanced ornithine decarboxylase expression
- Conversion of C57Bl/6 mice from a tumor promotion–resistant to a –sensitive phenotype by enhanced ornithine decarboxylase expression
- Alleviation of Multidrug Resistance by Flavonoid and Non-Flavonoid Compounds in Breast, Lung, Colorectal and Prostate Cancer
- The expression of the gene <i>asebia</i> in the laboratory mouse: 3. Sebaceous glands
- Abstract LB-039: Chronic inflammation-mediated contribution of bone marrow-derived epithelial cells and hair follicle stem cells to development of cutaneous neoplasms
- Abstract LB-039: Chronic inflammation-mediated contribution of bone marrow-derived epithelial cells and hair follicle stem cells to development of cutaneous neoplasms
- The human promyelocytic leukemia protein is a tumor suppressor for murine skin carcinogenesis
- Androgen Receptor Actions Modify Skin Structure and Chemical Carcinogen-induced Skin Cancer Susceptibility in Mice
- K15 promoter-driven enforced expression of NKIRAS exhibits tumor suppressive activity against the development of DMBA/TPA-induced skin tumors
- Nobiletin-loaded composite penetration enhancer vesicles restore the normal miRNA expression and the chief defence antioxidant levels in skin cancer
- Targeted disruption of Bcl‐x<sub>L</sub> in mouse keratinocytes inhibits both UVB‐ and chemically induced skin carcinogenesis
- Inducible Cutaneous Inflammation Reveals a Protumorigenic Role for Keratinocyte CXCR2 in Skin Carcinogenesis
- 137 Loss of myeloid Mcpip1 suppresses hair growth and development of epidermal papilloma of the skin
- Integrin α3β1 in hair bulge stem cells modulates CCN2 expression and promotes skin tumorigenesis
- Treatment with Light-Emitting Diodes of Wavelength 863 nm Delays DMBA/TPA-Induced Skin Tumor Formation and Decreases Proinflammatory Cytokine Levels in ICR Mice
- Author response: Loss of Dnmt3a and Dnmt3b does not affect epidermal homeostasis but promotes squamous transformation through PPAR-γ
- Promotion of Skin Carcinogenesis by Dimethylarsinic Acid in <i>Keratin (K6)/ODC</i> Transgenic Mice
- The 1,25-dihydroxyvitamin D3-independent actions of the vitamin D receptor in skin
- MCPIP1 controls effects of myeloid cells on skin carcinogenesis and hair growth
- Lgr6+ stem cells and their progeny in mouse epidermis under regimens of exogenous skin carcinogenesis, and their absence in ensuing skin tumors
- 1α,25(OH)2-Dihydroxyvitamin D3/VDR protects the skin from UVB-induced tumor formation by interacting with the β-catenin pathway
- GRowing an Epidermal Tumor
- Relation of Skin Polyamines to the Hairless Phenotype in Transgenic Mice Overexpressing Spermidine/Spermine N1-Acetyltransferase
- Cutaneous Two-Stage Chemical Carcinogenesis
- Decision letter: Loss of Dnmt3a and Dnmt3b does not affect epidermal homeostasis but promotes squamous transformation through PPAR-γ
- Impact of mTORC1 inhibition on keratinocyte proliferation during skin tumor promotion in wild‐type and BK5.Akt<sup>WT</sup> mice
- Context-dependent effect of sPLA2-IIA induced proliferation on murine hair follicle stem cells and human epithelial cancer
- Inactivation of the Vitamin D Receptor Enhances Susceptibility of Murine Skin to UV-Induced Tumorigenesis
- Chemically induced skin tumors arise from long-lived stem cells of the upper hair follicle