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Changes in keratin affect skin health and can lead to skin disorders like blistering diseases and psoriasis.

Keratins are important for skin cell health and their problems can cause diseases.

Activating c-Myc in skin causes rapid cell growth and changes, but these effects are reversible.

VDUP1 is found in skin and hair follicles, interacts with sciellin, and may help regulate skin cell differentiation.

Skin cell behavior is influenced by the tightness of nearby cells, affecting their growth and development.

Keratin genes change gradually during skin cell development and should be used carefully as biomarkers.

Keratin gene expression helps understand different types of skin cells and their development, and should be used carefully as biological markers.

Different Myc family proteins are located in various parts of the hair follicle and may affect stem cell behavior.

The document concludes that adult mammalian skin contains multiple stem cell populations with specific markers, important for understanding skin regeneration and related conditions.

The skin's internal clock affects healing, cancer risk, aging, immunity, and hair growth, and disruptions can harm skin health.

Keratin proteins are crucial for healthy skin, but mutations can cause skin disorders with no effective treatments yet.

Smad1 and Smad5 are essential for hair follicle development and stem cell sleepiness.

Stem cell differentiation is crucial for skin barrier maintenance and its disruption can lead to skin diseases.

Male pattern baldness may be caused by factors like poor blood circulation, scalp tension, stress, and hormonal imbalances, but the exact causes are still unclear.

Environmental cues can change the fate and function of epithelial cells, with potential for cell therapy.

Ovol2 is crucial for hair growth and skin healing by controlling cell movement and growth.

Keratin 15 expression in skin cells is regulated by two mechanisms involving PKC/AP-1 and FOXM1.

Retinoic acid-binding proteins in skin are regulated by β-catenin and Notch signalling.

Lack of Ctip2 in skin cells delays wound healing and disrupts hair follicle stem cell markers in mice.

Odontogenic keratocysts are caused by abnormal Hedgehog signaling and can lead to tooth and bone issues.

Caspases are enzymes important for both cell death and various non-lethal cell functions, affecting head development and hair growth, with different caspases playing specific roles.

Human skin can make serotonin and melatonin, which help protect and maintain it.

The document concludes that while significant progress has been made in understanding skin biology and stem cells, more research is needed to fully understand their interactions with their environment.

c-Myc activation in mouse skin increases sebaceous gland growth and affects hair follicle development.

A protein called desmoglein 3 is important for keeping hair follicle stem cells inactive and helps in their regeneration.

Epithelial stem cells can adapt and help in tissue repair and regeneration.

Epithelial stem cells are crucial for tissue renewal and repair, and understanding them could improve treatments for damage and cancer.

Sebaceous glands play a key role in skin health, immunity, and various skin diseases.

Epigenetic mechanisms control skin development by regulating gene expression.

Inhibiting AP1 in mice skin causes structural changes and weakens the skin barrier.