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The molecule Wise is involved in the development of various structures in chick embryos.

β-catenin in the dermal papilla is crucial for normal hair growth and repair.

The research shows that a gene called Wnt3 affects hair growth and structure, causing short hair and balding when overactive.

Ectodin integrates BMP, SHH, and FGF signals in developing ectodermal organs.

Hair follicle dermal stem cells are key for regenerating parts of the hair follicle and determining hair type.

The document concludes that a better understanding of cell changes during wound healing could improve treatments for chronic wounds and other conditions.

MIM is crucial for hair follicle formation and regeneration by controlling cilia formation and hedgehog signaling through its interaction with Cortactin and Src.

Mammals might fail to regenerate not because they lack the right cells, but because of how cells respond to their surroundings, and changing this environment could enhance regeneration.

The circadian clock influences the behavior and regeneration of stem cells in the body.

The nude gene causes skin cell overgrowth and improper development, leading to hair and urinary issues.

Msx2 and Foxn1 are both crucial for hair growth and health.

Dying cells can help with faster healing and new hair growth by releasing a growth-promoting molecule.

Dermal papillae cells, important for hair growth, come from multiple cell lines and can be formed by skin cells, regardless of their origin or hair cycle phase. These cells rarely divide, but their ability to shape tissue may contribute to their efficiency in inducing hair growth.

Ptprq has multiple forms that change during inner ear development.

Dermal EZH2 controls skin cell development and hair growth in mice.

The document concludes that while significant progress has been made in understanding skin biology and stem cells, more research is needed to fully understand their interactions with their environment.

Too much thymosin beta4 causes weird teeth and more hair growth in mice.

Inflammation plays a key role in activating skin stem cells for hair growth and wound healing, but more research is needed to understand how it directs cell behavior.

The review found that the way Platelet-Rich Plasma is made varies a lot, which can change the results of medical treatments.

Researchers created immortal human skin cells with constant testosterone receptor activity to study hair loss and test treatments.

Hair aging is caused by stress, hormones, inflammation, and DNA damage affecting hair growth and color.

The immune system plays a key role in tissue repair, affecting both healing quality and regenerative ability.

Different types of stem cells in hair follicles play unique roles in wound healing and hair growth, with some stem cells not originating from existing hair follicles but from non-hair follicle cells. WNT signaling and the Lhx2 factor are key in creating new hair follicles.

Stem cells in the hair follicle are regulated by their surrounding environment, which is important for hair growth.

The document concludes that understanding the sebaceous gland's development and function is key to addressing related skin diseases and aging effects.

Androgens can both stimulate and cause hair loss, and understanding their effects is key to treating hair disorders.

Genetic mutations can cause hair growth disorders by affecting key genes and signaling pathways.

Eph/ephrin signaling is important for skin cell behavior and could be targeted to treat skin diseases.

Gene network oscillations inside hair stem cells are key for hair growth regulation and could help treat hair loss.

Fisetin, a type of polyphenol, may help hair grow by increasing certain protein activities in cells.