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Fibroblast Growth Factors (FGFs) are important for tissue repair and regeneration, influencing cell behavior and other factors involved in healing, and are crucial in processes like wound healing, bone repair, and hair growth.

Skin's Regulatory T cells are crucial for maintaining skin health and could be targeted to treat immune-related skin diseases and cancer.

Activating TLR9 helps heal wounds and regrow hair by using specific immune cells.

Blood-derived CD34+ cells speed up healing, reduce scarring, and regrow hair in skin wounds.

The right cells and signals can potentially lead to scarless wound healing, with a mix of natural and external wound healing controllers possibly being the best way to achieve this.

A protein found in safflower seeds can stimulate hair growth and speed up wound healing in mice.

Blocking a protein called CXXC5 with a specific peptide can stimulate hair regrowth and new hair growth in wounds.

Hair loss can be treated with common methods like minoxidil and finasteride, but new potential treatments include growth factors, cytokines, and platelet-rich plasma injections.

Non-immune dermal cells dominate, epidermal cells increase after day 9, and certain immune cells persist beyond inflammation in wound-induced hair follicle regeneration.

Gamma delta T cells in the skin help with healing and defense but can also cause autoimmune issues, and more research is needed to understand how they are activated.

Blocking IL-17 signaling can delay skin aging and improve skin and hair health.

Imiquimod cream activates hair follicle stem cells and causes early hair growth by changing immune cells and certain protein expressions.

The skin has a complex immune system that is essential for protection and healing, requiring more research for better wound treatment.

Leptin-deficient mice, used as a model for Type 2 Diabetes, have delayed wound healing due to impaired contraction and other dysfunctional cellular responses.

Hair follicles are normally protected from the immune system, but when this protection fails, it can cause hair loss in alopecia areata.

Certain types of T cells are essential for healthy skin and play a role in skin diseases, but more research is needed to improve treatments.

Dermal stem cells help regenerate hair follicles and heal skin wounds.

Gene network oscillations inside hair stem cells are key for hair growth regulation and could help treat hair loss.

T-cell reconstitution after thymus transplantation can cause hair whitening and loss.

Targeting Vδ1+T-cells may help treat alopecia areata.

Jagged-1 in skin Tregs is crucial for timely wound healing by recruiting specific immune cells.

Special immune cells called Tregs can help prevent lung scarring by blocking a specific growth factor.

RANKL improves the immune response against herpes simplex virus by enhancing T cell activation and could help develop better treatments or vaccines.

Immune cells affect hair growth and could lead to new hair loss treatments.

Key genes linked to immune response are upregulated in hair follicles and skin tissues in chronic discoid lupus erythematosus.

Key genes linked to immune response are highly active in lupus-affected hair follicles.

Wound healing is complex and requires more research to enhance treatment methods.

The document concludes that more research is needed to better understand and treat primary cicatricial alopecias, and suggests a possible reclassification based on molecular pathways.

New understanding of the causes of primary cicatricial alopecia has led to better diagnosis and potential new treatments.

Alopecia areata causes hair loss due to an immune attack on hair follicles, influenced by genetics and environment.