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Dutasteride caused more penile tissue changes than finasteride, possibly increasing erectile dysfunction risk.

Oral finasteride and dutasteride may negatively affect erectile function in rats.

Biomaterials with MSC-derived substances could improve tissue repair and have advantages over direct cell therapy.

Unconventional lymphocytes are important for quick immune responses and healing of skin and mucosal barriers.

A brain-made hormone can protect against memory-related brain damage caused by harmful proteins.

Finasteride doesn't improve survival in LNCaP tumor model during off-cycle of therapy.

Mice with alopecia areata had wider lymphatic vessels in their skin.

Skin organoids are a promising new model for studying human skin development and testing treatments.

Low androgen levels might delay prostate cancer but could lead to more aggressive, therapy-resistant cancers.

The model showed that randomness accurately describes individual hair growth cycles and that synchronization can cause large fluctuations not seen in humans.

Valproic acid and rapamycin protect hair follicle stem cells from damage by activating a protective pathway.

Lichen Planopilaris and Frontal Fibrosing Alopecia may help us understand hair follicle stem cell disorders and suggest new treatments.

Restoring mitochondrial function in mice reversed their skin wrinkling and hair loss.

Flutamide, dutasteride, and finasteride dissolve differently in supercritical carbon dioxide, with dutasteride dissolving the least.

Effective treatments for spinal and bulbar muscular atrophy are not yet available; more research is needed.

Aging mice have slower hair regeneration due to changes in signal balance, but the environment, not stem cell loss, controls this, suggesting treatments could focus on environmental factors.

Removing Dicer from pigment cells in newborn mice causes early hair graying and changes in cell migration molecules.

Replenishing free 18-MEA can help restore damaged hair surfaces.

Blocking YAP/TAZ could be a new way to treat skin cancer.

Overexpressing SSAT enzyme reduces prostate tumor growth in mice.

Overexpressing SSAT in mice makes them highly sensitive to polyamine analogues, causing liver damage and high mortality.

ASH2L is essential for skin and hair development.

Androgens increase norepinephrine release, promoting smooth muscle growth in male sex organs, which may contribute to benign prostatic hypertrophy.

Retinoids or their analogs could treat skin pigmentation disorders like melasma and vitiligo.

Macrophages are vital for skin healing, hair growth, salt balance, and cancer defense.

Lymphocytes, especially CD8+ T cells, play a key role in causing alopecia areata, and targeting them may lead to new treatments.

The Ysc84/SH3yl1 protein family is important for cell movement and the process of taking in materials by interacting with actin and cell membranes.

Bacteria can help skin regenerate through a process called IL-1β signaling.

Neutrophil extracellular traps slow down hair follicle healing after injury.

A pathway helps maintain long telomeres in both stem and cancer cells.